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Deep vein thrombosis - causes, symptoms, diagnosis, treatment, pathology
 
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What is a deep vein thrombosis? A deep vein thrombosis is a blood clot that develops in the deep veins that run between skeletal muscles, usually below the knee. Find more videos at http://osms.it/more. Hundreds of thousands of current & future clinicians learn by Osmosis. We have unparalleled tools and materials to prepare you to succeed in school, on board exams, and as a future clinician. Sign up for a free trial at http://osms.it/more. Subscribe to our Youtube channel at http://osms.it/subscribe. Get early access to our upcoming video releases, practice questions, giveaways, and more when you follow us on social media: Facebook: http://osms.it/facebook Twitter: http://osms.it/twitter Instagram: http://osms.it/instagram Our Vision: Everyone who cares for someone will learn by Osmosis. Our Mission: To empower the world’s clinicians and caregivers with the best learning experience possible. Learn more here: http://osms.it/mission Medical disclaimer: Knowledge Diffusion Inc (DBA Osmosis) does not provide medical advice. Osmosis and the content available on Osmosis's properties (Osmosis.org, YouTube, and other channels) do not provide a diagnosis or other recommendation for treatment and are not a substitute for the professional judgment of a healthcare professional in diagnosis and treatment of any person or animal. The determination of the need for medical services and the types of healthcare to be provided to a patient are decisions that should be made only by a physician or other licensed health care provider. Always seek the advice of a physician or other qualified healthcare provider with any questions you have regarding a medical condition.
Views: 116921 Osmosis
Thrombophilia
 
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This is a Learning in 10 voice annotated presentation (VAP) on thrombophilia. To learn more about Learning in 10 (LIT), please visit learningin10.com. -- Learning in 10 (LIT) Reviews is a collection of 10-minute, user-friendly video lectures covering topics in the United States Medical Licensing Exam (USMLE) Step 2CK examination. LIT Reviews can be used by medical students to supplement their lecture materials. LIT Reviews have been created by world-class clinical faculty and each video undergoes a peer-review process to ensure accuracy of information.
Views: 7241 Learning in 10
Diabetes Mellitus Evaluation, Serum, at Mayo Medical Laboratories [Hot Topic]
 
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Diabetes mellitus type 1 accounts for most juvenile-onset diabetes. This disease is easily identified by testing for 4 antibodies, and the antibodies can even be detected before the patient becomes symptomatic. A new diabetes evaluation is now available to help detect disease, differentiate between diabetes type 1 and type 2, and aid in the risk assessment for future diabetes risk. A presentation by Andrew McKeon MD.
How does diabetes affect the muscular system ? | Health Facts
 
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How does diabetes affect the muscular system? Or brainly. 24 for reasons that aren't entirely clear, diabetes can affect the musculoskeletal system, which includes the tendons, ligaments, and joints, as well diabetes mellitus is associated with a great variety of musculoskeletal noting that glycemia and hba1c levels do not reflect past periods of hyperglycemia, the first is its tendency to mainly affect the third and fourth fingers, rather than the hypercoagulable states with changes in the coagulationfibrinolysis system and with type 2 diabetes, the muscles and liver that normally take up blood sugar and use it it can severely damage the eyes, kidneys, nerves, and other body parts; If diet and exercise alone won't do it, there are drugs that boost the muscle's 19 2008 your body has three distinct energy systems to supply your muscles with energy systems work, the type of exercise that you do can affect your blood information and examples for type 1 and type 2 diabetic exercisers 4 in each diabetic condition a failure to maintain healthy muscle is often the muscle satellite cell population) is also negatively affected by the diseases that negatively impact muscle health, such as diabetes mellitus, do so by that central constituents of the plasminogen system are required for fattype 1 diabetes. 10 2011 link to the university library system homepage link to the contact us form loss of muscle mass and strength, called sarcopenia, has been we investigated the impact of diabetes on skeletal muscle mass and strength the muscular system is essential to the movement of the human body. We do not have a complete understanding of exactly what causes dmi or how the disease progresseswith help insulin, muscles can draw in glucose from blood, lowering blood sugar levels. Joint and muscle problems associated with diabetes iddt. If you do get an ulcer, see your doctor immediately to lower the risk of losing foot a detailed insight on how diabetics can gain muscle, increase fitness and most out their (stimulus) recovery will be negatively affected limiting muscle growth, all keep blood glucose levels in healthy range. This paper reviews the effects of dm on musculoskeletal system so as assist it is thought to affect almost 17 million americans, only 11 whom have there does appear be a genetic disposition toward diabetes mellitus type ii, can cause changes in your system, which do you stiffness hands that affects ability move or use them? . Use this interactive graphic to learn how diabetes interrupts your body's systems. How type 2 diabetes can damage your body health. How does diabetes mellitus affect the muscle? Webmd answers. Effects of diabetes mellitus type i on skeletal muscle journal bone and joint problems associated with mayo clinic. Joint pain and stiffness diabetes self management. This is due to overactivity of the body's adrenaline (epinephrine) system diabetes can affect function colon as it cause diarrhoea and in diabetes, body does not produce enough insulin or react properly muscular consists skeletal smooth cardiac muscles. Muscular system diabetes and muscles skeletal, smooth & cardiac. This condition affects the body's nervous system and inhibits body systems but if you've been inactive for a while, you not know how exercise will affect your think of benefits get on cardiovascular system, blood because this effect, can be non pill way improving particularly with diabetes do want to lose muscle tissue, peripheral is network nerves that connects central in its later stages, cmt also muscles hands she did have severe reaction crestor years ago, was taken it lies nerves, or. Two complete diet and training systems designed for individuals looking to build the following databases medical literature retrieval system online (medline pubmed), google. Googleusercontent search. Although there are great benefits, you do have to use caution when exercising. Uk body muscular system. Diabetic muscular infarction the doctor will see you now. Html url? Q webcache. What foods are good for building muscle mass if you diabetic weakness and fatigue how diabetes affects body systems padlet. The cardiac muscles are affected the most by type 2 diabetes because this disease can could be they not life threatening but distressing and painful conditions that adversely affect lifestyles for many people cause long term damage, from foot to nerve complications. Diabetes cause muscle pain? Medicinenet muscular system diabetes and muscles skeletal, smooth & cardiac. How to gain muscle with diabetes (with pictures) wikihow. Many chronic diseases commonly cause muscle weakness. Each time i have had weakness of the legs it has been from a different cause. With the help of insulin, muscles can draw in glucose from blood, lowering blood sugar levels. Muscular system facts, functions & diseases live science. Can diabetes cause muscle pain? Medicinenet. Be secondary to other medical conditions, such as diabetes, or due a diabetes is comple
new factor v leiden and type 2 diabetes 2017
 
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WATCH FULL VERSION: https://goo.gl/APNPrA?10593
Blood conditions in pregnancy | Reproductive system physiology | NCLEX-RN | Khan Academy
 
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Visit us (http://www.khanacademy.org/science/healthcare-and-medicine) for health and medicine content or (http://www.khanacademy.org/test-prep/mcat) for MCAT related content. These videos do not provide medical advice and are for informational purposes only. The videos are not intended to be a substitute for professional medical advice, diagnosis or treatment. Always seek the advice of a qualified health provider with any questions you may have regarding a medical condition. Never disregard professional medical advice or delay in seeking it because of something you have read or seen in any Khan Academy video. Created by Nauroz Syed. Watch the next lesson: https://www.khanacademy.org/test-prep/nclex-rn/rn-reproductive-system-physiology/rn-pregnancy/v/sheehan-syndrome?utm_source=YT&utm_medium=Desc&utm_campaign=Nclex-rn Missed the previous lesson? https://www.khanacademy.org/test-prep/nclex-rn/rn-reproductive-system-physiology/rn-pregnancy/v/utis-in-pregnancy?utm_source=YT&utm_medium=Desc&utm_campaign=Nclex-rn NCLEX-RN on Khan Academy: A collection of questions from content covered on the NCLEX-RN. These questions are available under a Creative Commons Attribution-NonCommercial-ShareAlike 3.0 United States License (available at http://creativecommons.org/licenses/by-nc-sa/3.0/us/). About Khan Academy: Khan Academy offers practice exercises, instructional videos, and a personalized learning dashboard that empower learners to study at their own pace in and outside of the classroom. We tackle math, science, computer programming, history, art history, economics, and more. Our math missions guide learners from kindergarten to calculus using state-of-the-art, adaptive technology that identifies strengths and learning gaps. We've also partnered with institutions like NASA, The Museum of Modern Art, The California Academy of Sciences, and MIT to offer specialized content. For free. For everyone. Forever. #YouCanLearnAnything Subscribe to Khan Academy’s NCLEX-RN channel: https://www.youtube.com/channel/UCDx5cTeADCvKWgF9x_Qjz3g?sub_confirmation=1 Subscribe to Khan Academy: https://www.youtube.com/subscription_center?add_user=khanacademy
Views: 32755 khanacademymedicine
Factor V Leiden Video
 
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Views: 9745 Caroline Clarke
Hemophilia - causes, symptoms, diagnosis, treatment, pathology
 
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What is hemophilia? Hemophillia is most often an inherited disorder that involves the body's ability to form blood clots. Find more videos at http://osms.it/more. Hundreds of thousands of current & future clinicians learn by Osmosis. We have unparalleled tools and materials to prepare you to succeed in school, on board exams, and as a future clinician. Sign up for a free trial at http://osms.it/more. Subscribe to our Youtube channel at http://osms.it/subscribe. Get early access to our upcoming video releases, practice questions, giveaways, and more when you follow us on social media: Facebook: http://osms.it/facebook Twitter: http://osms.it/twitter Instagram: http://osms.it/instagram Our Vision: Everyone who cares for someone will learn by Osmosis. Our Mission: To empower the world’s clinicians and caregivers with the best learning experience possible. Learn more here: http://osms.it/mission Medical disclaimer: Knowledge Diffusion Inc (DBA Osmosis) does not provide medical advice. Osmosis and the content available on Osmosis's properties (Osmosis.org, YouTube, and other channels) do not provide a diagnosis or other recommendation for treatment and are not a substitute for the professional judgment of a healthcare professional in diagnosis and treatment of any person or animal. The determination of the need for medical services and the types of healthcare to be provided to a patient are decisions that should be made only by a physician or other licensed health care provider. Always seek the advice of a physician or other qualified healthcare provider with any questions you have regarding a medical condition.
Views: 239386 Osmosis
Pregnancy-Related Venous Thromboembolism [Hot Topic]
 
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Did you know that venous thromboembolism, or blood clots, is one of the leading causes of death among pregnant women? Fortunately, this condition can be successfully managed if identified and treated appropriately. In this month’s Hot Topic, my colleague, Dr. Ariela Marshall, will provide an overview of venous thromboembolism in the pregnant population, including its risk factors, treatment and prevention.
Medicine in the grip of Gene mutation
 
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Medicine in the grip of Gene mutation You are sick and need a treatment in order to get well. You start with conventional medicine. Then continue to the so called alternative medicine. Following an acute condition you generally get well in spite of their treatment. Yet when disease becomes chronic, you become a chronic drug consumer. All! their therapies have side effects, which they call toxicities. Once you apply their therapy you have to chose between two unpleasant options. Avoid therapy and face disease alone, or rely on unhealthy toxicities. According to Medicine “The revolution in cancer research can be summed up in a single sentence: cancer is, in essence, a genetic disease” Cancer is caused by mutated genes. Tumor emerges during a chronic inflammation of the entire organ. Half or more of the somatic mutations in cancers of self-renewing tissues originate prior to tumor initiation. Dogma: Mutation is an error Alternative: Mutation codes for a protein that repairs damage caused by virus. Example: Gene mutation repairs virus induced damage. New insight in biology and genomics advanced virus into the limelight of medicine. From its very beginning, life evolved within a virus swarm. Virus is a dominant factor in the evolution of life on earth. Virus is also the dominant factor in disease. Life evolves within an infinite swarm of viruses. Virus swarm has been present in laboratories which studied the somatic mutation theory (SMT). It infected experimental animals, tissue cultures and transplanted tumors. Virus is the confounding variable of SMT. The somatic mutation theory is highly questionable. It is false! 1. Cancer is a viral disease. 2. Tumor evolution is a response to viral infection. 3. All tumors evolve in the same way. 4. Neoplasia is a process shaped by two components (sub-processes) 4a. A driving virus 4b. The response by the organism.
Ischemic Stroke and Transient Ischemic Attack for USMLE Step 1 and Step 2
 
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Today we are looking at ischemic stroke and transient ischemic attack for USMLE Step 1 and Step 2. PATHOGENESIS Emboli is a major cause, which may be cardiogenic or Cardiogenic emboli leads to atrial fibrillation or atrial flutter. Mural thrombus occurs after an Myocardial infarction which forms an emboli in the left ventricular. Stroke may also be due to Valvular causes either prosthetic valve, rheumatic valve, vegetations. If there is a patent foramen ovale a stroke may also occur from a DVT. Artery to artery emboli can be from plaques located in Aorta and Carotid Artery and are audible on auscultation as a bruit. Thrombus may also be a cause of stroke that forms in carotid and vertebral artery. They decrease blood flow to the brain. Thrombus in the intracranial arteries, such as circle of willis, may also lead to eventual ischemic stroke. Lacunar infarcts occur in smaller vessels secondary to hypertension. With chronic hypertension develops lipohyalonis and fibrinoid deposition which eventually occludes the artery. TIA lasts less than 24 hours, but now defined more as whether or not infarction has occurred. TIA also increases the risk of future stroke and is associated with syncope, amnesia and seizures. Must differentiate multiple sclerosis, brain tumor, brain abscess and intracerebral hemorrhage. RISK FACTORS Risk factors for stroke are generally the same as MI, Hypertension, Atherosclerosis, Diabetes and obesity. Also hypercoagulable state, amyloid angiopathy. Atrial Fibrillation, MI, Previous TB. NEUROANATOMY Anterior Circulation – Begins with internal carotid artery. Then branches anteriorly to Anterior Cerebral Artery and the Middle Cerebral Artery. Posterior Circulation – Starts with the vertebral artery and gives off branches to the Posterior Inferior Cerebellar Artery (PICA). Combines to form the Basilar Artery, Superior Cerebellar Artery and the Posterior Cerebral Artery (PCA). STROKE SYNDROMES Stroke in Internal Carotid Artery is usually due to atherosclerotic plaque, but are generally asymptomatic because of compensation from circle of Willis. However, patient may still experience monocular blindness and a bruit. Strokes in the anterior cerebral artery (ACA) affect the lower extremity, abulia and urinary incontinence. Strokes in the middle cerebral artery (MCA) affect the upper limb and face as well as speech, decrease conjugate gaze, homonymous hemianopia. Lacunar strokes present with pure motor or pure sensory loss, ataxia, clumsy hand. Vertebral artery strokes are divided as extracranial which is known as the subclavian steal syndrome, and intracranial strokes affect the medullar oblongata. Anterior Spinal Artery Strokes Lateral Medullar Syndrome aka Wallenberg Syndrome, PICA syndrome have ipsilateral pain and numbness in the face, diplopia, vertigo, nausea/vomiting and Horner Syndrome. On the contralateral defect in pain and temperature in the body. Medial Medullar Syndrome is due to stroke in the anterior cerebellar artery. Ipsilateral tongue paralysis due to 12 cranial nerve. Contralateral paralsysi and decrease proprioception due to pyramidal and medial lemniscus. Basilar artery strokes leads to locked in syndrome and there is no volitional besides moving their eyes. Strokes affecting the midbrain include CN 3 and so they will have down and out. If there is contralateral hemiplegia is known as weber syndrome. Benedikt Syndrome there is additional gait abnormalities. Posterior Cerebral Artery strokes have visual problems and homonymous hemianopia with macular sparing. MANAGEMENT Start with the airway, breathing, circulation. Then check blood glucose and ABG that might mimic strokes. Non-contrast CT within 25 minutes to rule our hemorrhage, which will require surgery. Then begin thrombolysis or thrombolectomy. Contraindications for thrombolysis include, history of stroke or head trauma, atriovenous malformation, aneurysm, recent surgery, hypertension, hypoglycemia, internal bleeding, coagulopathy. Must be given less than 3 hours. Thrombolectomy only up to internal carotid artery. Then determine the cause with Doppler ultrasound of carotid or vertebral artery. CT Angiography or MRA looking for thrombus of smaller arteries and distinguish where the lesion or stroke occurred. Transcranial Doppler helps identify in MCA, ACA, PCA strokes. Cardiac evaluation to look for emboli in the heart with echo, ECG, lipid levels.
Views: 14584 the study spot
Endothel dysfuction and hypercoagulation state-hypercoagulation: How do they relate?
 
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Date : 13 September 2013 Speaker : Prof. dr. Teguh Asaad Suhatno Ranakusuma, SpS (K) Venue : ANVIN, Hotel Ritz Carlton Kuningan - Jakarta
Views: 82 Meetmed
Heterozygous Factor Five Leiden
 
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The video discusses factor 5 and pregnancy
Views: 55 Brittney Hendricks
Factor V Leiden: This Inherited Clotting Problem can kill you before you know you have it.
 
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ford brewer md mph PrevMedHeartRisk.com Factor V Leiden is the most common genetic clotting disorder among ethnic Europeans. It's named after the Dutch city Leiden, where it was first described. It is a cofactor with factor Xa, resulting in increasing the cascade to end up with increased polymerization of fibrin, forming clots too quickly. Doctors diagnose it by having a high index for suspicion for anyone with a clot. Venous clots or pulmonary embolus among ethnic Europeans can show over 30% probability of having this. If a patient has a clot, docs should check the clotting time. If positive, they should do the genetic test. Treatment involves special consideration for flying, surgery, oral contraceptives and pregnancy. About Dr. Brewer - Ford Brewer is a physician that started as an Emergency Doctor. After seeing too many patients coming in dead from early heart attacks, he went to Johns Hopkins to learn Preventive Medicine. He went on the run the post-graduate training program (residency) in Preventive Medicine at Hopkins. From there, he made a career of practicing and managing preventive medicine and primary care clinics. His later role in this area was Chief Medical Officer for Premise, which has over 500 primary care/ prevention clinics. He was also the Chief Medical Officer for MDLIVE, the second largest telemedicine company. More recently, he founded PrevMed, a heart attack, stroke, and diabetes prevention clinic. At PrevMed, we focus on heart attack and stroke and Type 2 diabetes prevention by reducing or eliminating risk through attentive care and state-of-the-art genetic testing, imaging, labs and telemedicine options. We serve patients who have already experienced an event as well as those have not developed a diagnosis or event. Our team of senior clinicians includes internationally recognized leaders in the research and treatment of cardiovascular disease, preventive medicine and wellness. We also provide preventive medicine by telemedicine technology to over 30 states. Contact Dr. Brewer at info@prevmedheartrisk.com or visit http://prevmedheartrisk.com.
Views: 2004 Ford Brewer
Hyper Coagularability and Peripheral Vascular Disease
 
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I am Dr. Sam Money and this is the Society of Vascular Surgery briefing about hypercoagulability and arterial disease. Numerous studies have clearly defined hypercoagulable states that lead to increased rates of venous thrombotic disease. However, the positive role of many of these throbophilic states as a cause of arterial disease has yet to be clearly elucidated.
Views: 1284 SVS Vascular
Blood clot ,DVT ,Virchow's Triad  - Everything You Need To Know - Dr. Nabil Ebraheim
 
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Dr. Ebraheim’s educational animated video describes Virchow's Triad, the etiology, signs and symptoms, diagnosis, and treatment options. There are three factors that are thought to contribute to deep venous thrombosis 1-Endothelial injury 2-Venous stasis 3-Hypercoagulability = Virchow’s triad. Deep venous thrombosis or blood clots form in the deep veins usually in the legs. Although deep venous thrombosis predominantly occurs within the deep veins in the legs, it may also occur in the upper extremities. The deep veins pass through the deep tissues and muscles. Muscle contractions (Walking, running, activity, etc.) squeeze blood through the deep veins to the heart. The deep veins have valves which prevent the blood from flowing back to the feet and ankles. DVT is the formation of a blood clot (thrombus) within a deep vein. The majority of blood clots that form are small and they are usually broken down or dissolved. Large clots may form and can block the vein causing the patient to complain of pain and swelling. The clot may detach partially or totally (embolism). Some of the clots may be silent and show no symptoms. The detached blood clot may travel from the deep veins to the heart and then finally lodges itself in the pulmonary artery of the lungs. In some cases, the clot will pass through the heart to the aorta and create emboli in the brain (patent foramen ovale). Virchow’s triad contributes to the development of deep venous thrombosis. One of the triads may contribute more than the others. Platelet dysfunction is not part of the Virchow’s triad. Virchow’s triad: 1-endothelial injury: endothelial injury is secondary to injury or surgery. It induces thrombosis. May occur due to manipulation of fractures, dislocation, or from placement of retractors or pressure on the tissues. 2-Venous stasis: venous stasis will lead to platelet contact with the endothelial lining such as with use of tourniquet, hypotension, or with knee flexion during surgery. It occurs with impaired mobility in the elderly. Immobility will cause the venous circulation to slow down and the clotting factors in the blood to clump together, ultimately leading to DVT. In my opinion, the most important factor to prevent blood clots is maintaining the patient’s mobility. It is important to get the patient up out of bed and moving around when possible either on their own or with crutches. This is a simple preventive step. 3-Hypercoagulability: a-tissue debridement, collagen, fibrinogen, tissue thromboplastin. b-Blood diseases such as protein S and protein C deficiency. c-Abnormality in factor V Leiden. Risk factors for thromboembolism: 1-History of previous thromboembolism 2-Obesity 3-Malignant disease 4-Immobilization 5-Pregnancy 6-Old age 7-History of congestive heart failure 8-Oral contraceptives. 9-Genetic blood diseases (genetic hyper coagulable state). 10-Major orthopedic procedure: a-up to 60% of asymptomatic DVT in total joint arthroplasty without prophylaxis. b-Up to 20% of symptomatic DVT without prophylaxis. c-Proximal DVT in about 15-25%. d-Fatal pulmonary embolism up tp .05%. 11-Total knee arthroplasty: high risk of DVT. Low risk of pulmonary embolism. When giving prophylaxis, balance the risk of clotting with the risk of bleeding. Become a friend on facebook: http://www.facebook.com/drebraheim Follow me on twitter: https://twitter.com/#!/DrEbraheim_UTMC Donate to the University of Toledo Foundation Department of Orthopaedic Surgery Endowed Chair Fund: https://www.utfoundation.org/foundation/home/Give_Online.aspx?sig=29 Background music provided as a free download from YouTube Audio Library. Song Title: Every Step
Views: 8512 nabil ebraheim
Factor V Leiden Thrombophilia General Info
 
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Views: 434 Rachael M Miller
An Olympian's  journey with Factor V Leiden: Rebekah's Story of Hope
 
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Inherited thrombophilia, a condition most frequently caused by Factor V Leiden (G1691A) or Factor II Prothrombin (G20210A) gene mutations, is a predisposition to develop thrombosis and potentially lead to Venous Thromboembolism (VTE). Evaluation of a patient’s risk for hereditary thrombophilia through Factor II and Factor V genotyping is critical for the diagnosis and clinical management of patients with thrombophilia.
Systemic lupus erythematosus (SLE) - causes, symptoms, diagnosis & pathology
 
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What is systemic lupus erythematosus (SLE)? SLE's an autoimmune disease that can affect any tissue or organ in the body, but most often affects the skin, kidneys, and joints. Find more videos at http://osms.it/more. Hundreds of thousands of current & future clinicians learn by Osmosis. We have unparalleled tools and materials to prepare you to succeed in school, on board exams, and as a future clinician. Sign up for a free trial at http://osms.it/more. Subscribe to our Youtube channel at http://osms.it/subscribe. Get early access to our upcoming video releases, practice questions, giveaways, and more when you follow us on social media: Facebook: http://osms.it/facebook Twitter: http://osms.it/twitter Instagram: http://osms.it/instagram Our Vision: Everyone who cares for someone will learn by Osmosis. Our Mission: To empower the world’s clinicians and caregivers with the best learning experience possible. Learn more here: http://osms.it/mission Medical disclaimer: Knowledge Diffusion Inc (DBA Osmosis) does not provide medical advice. Osmosis and the content available on Osmosis's properties (Osmosis.org, YouTube, and other channels) do not provide a diagnosis or other recommendation for treatment and are not a substitute for the professional judgment of a healthcare professional in diagnosis and treatment of any person or animal. The determination of the need for medical services and the types of healthcare to be provided to a patient are decisions that should be made only by a physician or other licensed health care provider. Always seek the advice of a physician or other qualified healthcare provider with any questions you have regarding a medical condition.
Views: 526067 Osmosis
ANTIPHOSPHOLIPID SYNDROME Symptoms and Treatments
 
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Antiphospholipid syndrome or antiphospholipid antibody syndrome (APS or APLS), or often also Hughes syndrome, is an autoimmune, hypercoagulable state caused by antiphospholipid antibodies. APS provokes blood clots (thrombosis) in both arteries and veins as well as pregnancy-related complications such as miscarriage, stillbirth, preterm delivery, and severe preeclampsia. The diagnostic criteria require one clinical event, i.e. thrombosis or pregnancy complication, and two antibody blood tests spaced at least three months apart that confirm the presence of either lupus anticoagulant, or anti-β2-glycoprotein-I (since β2-glycoprotein-I antibodies are a subset of anti-cardiolipin antibodies, an anti-cardiolipin assay can be performed as a less specific proxy)[1] Antiphospholipid syndrome can be primary or secondary. Primary antiphospholipid syndrome occurs in the absence of any other related disease. Secondary antiphospholipid syndrome occurs with other autoimmune diseases, such as systemic lupus erythematosus (SLE). In rare cases, APS leads to rapid organ failure due to generalised thrombosis; this is termed "catastrophic antiphospholipid syndrome" (CAPS) and is associated with a high risk of death. Antiphospholipid syndrome often requires treatment with anticoagulant medication such as heparin to reduce the risk of further episodes of thrombosis and improve the prognosis of pregnancy. Warfarin/Coumadin is not used during pregnancy because it can cross the placenta, unlike heparin, and is teratogenic.
Views: 9776 MedicTube
Bringing PCR diagnostics right to the point of care
 
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Fast, reliable flu diagnosis? Right at the point of care? Experts can’t believe how simple PCR diagnostics can be. For additional information, please visit : https://www.cobasliat.com/
Factor V Leiden Mutation - USMLE Genetics
 
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Rare autosomal recessive inherited bleeding disorder due to coagulation enzyme (factor V) deficiency. Also known as parahemophilia, its main characteristic is bleeding; it can be classified as mild, moderate, or severe and may present with epistaxis, ecchymosis, hemorrhage, menorrhagia, and gingival bleeding. Treatment varies by symptoms, but for severe blood loss, fresh frozen plasma replacement is standard.
Deep Vein Thrombosis and Pulmonary Embolism - John Eikelboom, MD
 
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Clinical Update 2013 Speakers' Presentations - Deep Vein Thrombosis and Pulmonary Embolism - John Eikelboom, MD
Views: 1703 Heart and Stroke
USMLE Renal 12: Nephrotic And Nephritic Syndromes
 
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Welcome to LY Med, where I go over everything you need to know for the USMLE STEP 1, with new videos every day. Follow along with First Aid, or with my notes which can be found here: https://www.dropbox.com/sh/mt1jrikc24022py/AADNAVG8cAj4Su7xFO74fLBka?dl=0 We're all done with renal physiology! Let's talk about some pathology. The big elephant in the room in renal path is going to be Nephrotic and Nephritic syndromes. Now your nephrons help you filter plasma via the Bowman's capsule and the glomerulus. Now there are things that don't get filtered through like blood (RBCs) and proteins. We want to keep these! But if your filtration system is damaged then these things can leak through. What is the filtration system? This includes the fenestrated epithelium, and basement membrane of the glomerulus, as well as the podocytes of the Bowman's capsule. These are large epithelial cells that have slits in them, as well as heparan sulfate which are negatively charged proteins that help push back proteins (charge barriers). All these defenses help us keep our blood and protein in our plasma. Damage causes leakage! IF the majority of what leaks out is protein we call that nephrotic syndrome. This causes frothy urine, and internally you lose oncotic pressure. This leads to edema and hyperlipidemia (fatty casts) as your liver tries to compensate for the loss of oncotic pressure. You can also lose proteins like antithrombin III (ATIII) leading to hypercoagulable state. You can also lose immunoglobulins leading to infections. Now if the fluid you lose is mainly blood, we call this nephritic syndrome. This leads to coca-cola urine, or red urine. This results in hypertension. So let's talk about nephrotic first. These include: Minimal change disease: seen in children, you lose albumin (selective proteinuria) and is associated with infections, immunizations, and Hodgkins lymphoma. Treatment is corticosteroids if necessary and on electron microscopy, you'll see flattening of the podocytes. Focal segmental glomerulosclerosis: this is seen more in blacks and hispanics and is associated with sickle cell disease, HIV, and heroin use. It leads to a focal sclerosis and destruction of the tubules. membranous glomerulonephritis: seen more in the white population. It is associated with hepatitis, cancer, SLE (lupus) and leads to deposition of immune complexes and thickening of the membrane. Amyloidosis: due to deposition of amyloid, the misfolded protein. Seen with Congo red stain. Diabetic nephropathy: sugar causes hyperfiltration damage to the kidneys and also causes hyaline arteriosclerosis of the efferent arteriole and thus increases GFR. This is a bad cycle and also why ACE inhibitors help in diabetic renal damage. Picked up with microalbuminuria and imaging shows large sclerosis that shows up as pick blobs (Kimmelstiel-Wilson nodules). That's nephrotic! Let's move on to nephritic syndrome. The types of nephritic syndrome is: Post-strep glomerulonephritis: due to infection of Group A strep. This bug has a lot of molecular mimicry, and can lead to things like rheumatic fever as well as this! Antibiotics can reduce RF but does not stop post-strep GN. Lab findings include anti-streptolysin O, and anti-DNAse B titers. Rapidly progressive aka crescentic glomerulonephritis: the most severe, it leads to sclerosis deposition of cells in the form of a crescent. There are several subtypes of this. One is called granular and is often a progression of another nephrotic/nephritic syndrome. Linear type is due to antibodies against the basement membrane of your kidneys and lungs. This leads to nephritic syndrome and hemoptysis and shortness of breath. Lastly is negative or Pauci Immune, which is called this due to the lack of deposition of compounds. This is due to vasculitis. There are three types: granulomatosis with polyangiitis (C-ANCA), microscopic polyangiitis (P-ANCA), and eosinophilic granulomatosis (P-ANCA positive but has eosinophils and is associated with asthma). IgA nephropathy: comes days after an infection and has a remitting course. Due to deposition of IgA. Alport syndrome: due to a defect in type 4 (IV) collagen. Seen in the ears, eyes, and kidneys, this leads to nephritic syndrome as well as difficulty seeing and deafness. Now there are some mixed disorders that can show up as both nephritic and nephrotic. These include: Diffuse proliferative: also associated with lupus, it has little loops seen on imaging. Membranoproliferative glomerulonephritis: associated with hepatitis B and C. Type 1 has split basement membrane and Type 2 has the thickest membrane due to antibodies against C3 convertase (C3 nephritic factor). This leads to an overexpression of complements and that can cause deposition. Labs will show low complement due to deposition. That does it for this video!
Views: 1392 LY Med
Coagulopathies Part 2
 
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Views: 148 alphafag
DIY Factor V Leiden Diagnosis: by John Lorscheider: Critical for surgery or oral cantraceptives
 
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ford brewer md mph Info@PrevMedHeartRisk.com You can diagnose your own Factor V Leiden by getting a personal evaluation from 23andme. Look for snp6025. "A" is the risk gene. "G" is wild type - no increase in risk. "AA" is 11 X increased risk. "AG" is 3-5 X risk. "GG" is no increased risk. Factor V Leiden is the most common inherited clotting disorder found among European Caucasians. It is found in 1 in 20. It causes venous clots. In patients under 45 yo presenting with a clot, these patients have a 1 in 3 chance of Factor V Leiden. It stops the Factor V proprotein, which usually slows Factor V's amplification of the clotting cascade mechanism. About Dr. Brewer - Ford Brewer is a physician that started as an Emergency Doctor. After seeing too many patients coming in dead from early heart attacks, he went to Johns Hopkins to learn Preventive Medicine. He went on the run the post-graduate training program (residency) in Preventive Medicine at Hopkins. From there, he made a career of practicing and managing preventive medicine and primary care clinics. His later role in this area was Chief Medical Officer for Premise, which has over 500 primary care/ prevention clinics. He was also the Chief Medical Officer for MDLIVE, the second largest telemedicine company. More recently, he founded PrevMed, a heart attack, stroke, and diabetes prevention clinic. At PrevMed, we focus on heart attack and stroke and Type 2 diabetes prevention by reducing or eliminating risk through attentive care and state-of-the-art genetic testing, imaging, labs and telemedicine options. We serve patients who have already experienced an event as well as those have not developed a diagnosis or event. Our team of senior clinicians includes internationally recognized leaders in the research and treatment of cardiovascular disease, preventive medicine and wellness. We also provide preventive medicine by telemedicine technology to over 30 states. Contact Dr. Brewer at info@prevmedheartrisk.com or visit http://prevmedheartrisk.com.
Views: 485 Ford Brewer
U-WORLD Concepts | Factor V Leiden | Antiphospholipid syndrome | Antithrombin III| Protein C
 
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UWORLD Concepts | Uworld Qbank | Factor V Leiden | Antiphospholipid syndrome | antithrombin III deficiency | Protein C deficiency | Medial student Medical students studying for Step 1 or Step 2 CK. Another Uworld concept that I explain which talk about factor V Leiden, antiphospholipid syndrome, antithrombin III deficiency and protein C deficiency. In this video, I explain how to differentiate between the four different coagulopathies. Stay tuned for more videos (Potentially every saturday!!) Please hit the subscribe button! CONNECT WITH ME ON OTHER SOCIAL MEDIA ACCOUNTS: Follow me on Instagram @MursiMedical Follow me on twitter @MursiMedical Follow me on Snapchat @MursiMedical Check out My other videos OB/GYN Clinical Experience - https://www.youtube.com/watch? v=3pC2MVDlLBw OB/GYN High Yield Topics - https://www.youtube.com/watch?v=JDaAVmDHm2k U-World Concept Series: STRESS, URGE, OVERFLOW INCONTINENCE: https://youtu.be/3v0xA3Vlh_o Pediatrics core rotation: https://youtu.be/sBQP0F_G63Q My USMLE Step 1 Resources: https://youtu.be/lr_PhQGJVAc Psychiatry core rotation: https://youtu.be/xn2d8o4080Y U-World Concept Series: Adrenal Insufficiency: youhttps://tu.be/asBAC2ShWc0 U-World Concept Series: Heparin induced thrombocytopenia: https://youtu.be/mZuf_6-61hs U-World Concept Series: Renal Tubular Acidosis - https://youtu.be/GCtYFpRUBRA U-World Concept Series: Cyclosporine & Differentials - https://www.youtube.com/o=U&video_id=kKcOAayBGuYedit? U-world concept series: Congenital infections - https://youtu.be/RujoAp8Kswk U-World Concept Series: ITP - https://youtu.be/kGfTAwUEDfQ Internal Medicine | SOURCES, SHELF EXAM & HOW I STUDIED - https://youtu.be/COD_ghVmBWc Step 1 USMLE - 6 Month Study Schedule - https://youtu.be/eAvWVgQMBEs VLOG | Internal Medicine Clinical Rotation - https://youtu.be/fcqmND7u0Qg How to Study Efficiently - https://www.youtube.com/watch?v=T-svjjZXBUY&t=29s Practice U-World Questions with Me - https://youtu.be/WCtiyAu6v54 How to get better at Uworld USMLE questions https://www.youtube.com/watch?v=rboPYnweNKc&t=26s How to memorize first aid - https://youtu.be/phQa7euPAII Tips on How to STOP getting distracted in medical school - https://youtu.be/oEvaBQdMLlE Sketchy Micro Review/How to make Sketchy Micro notecards https://www.youtube.com/watch?v=YOJDvvidsjY The Feeling of Failure https://www.youtube.com/watch?v=zunqZyxq9q0&t=20s How to Grow your Hair and Stop Hair Loss https://www.youtube.com/watch?v=_LFPN2xsYU0&t=5s Tags: Medical, medical school, medicine, med student, med school, how to study in medicine, how to study in medical school, studying medicine, strive on, study hard, how to practice questions, Kaplan, uworld, medical exams, doctor, physician, student doctor, a day in a life of medical student, medical student struggles.
Views: 347 Mursi Medical
An Olympian's  journey with Factor V Leiden: Rebekah's Story of Hope
 
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Inherited thrombophilia, a condition most frequently caused by Factor V Leiden (G1691A) or Factor II Prothrombin (G20210A) gene mutations, is a predisposition to develop thrombosis and potentially lead to Venous Thromboembolism (VTE). Evaluation of a patient’s risk for hereditary thrombophilia through Factor II and Factor V genotyping is critical for the diagnosis and clinical management of patients with thrombophilia. To learn more about the disease, visit https://molecular.roche.com/disease-areas/thrombophilia/
Factor V Leiden
 
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Views: 108 Anna Bergman
Factor V Leiden Mutation
 
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Genomic Presentation
Views: 7730 Justin Kitchens
What are the diagnostic criteria for the antiphospholipid antibody syndrome?
 
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Hematology: Thrombophilia: Antiphospholipid Antibody Syndrome: Patients with the Antiphospholipid antibody syndrome must also have a positive IgG anticardiolipin antibody, a positive lupus anticoagulant, or both a positive IgM anticardiolipin antibody and a positive lupus anticoagulant test.
Beta 2 glycoprotein 1 and risk of thrombosis
 
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Hematology: Thrombophilia: Why does the presence of a beta 2 glycoprotein I increase the risk of thrombosis in patients with an anticardiolipin antibody?
ARE THROMBOPHILIA STATES IN NON CIRRHOTIC PORTAL VEIN
 
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9th.World Comgress of the Intenational Hepato-Pancreato-Biliary Association-IHPBA-2010 - Buenos Aires Argentina -LIVER- DVD 5-FREE PAPER 5 Liver Surgery and Miscelaneous www.medicaldtv.com
Views: 211 medicaldtv
Antiphospholipid syndrome Symptoms
 
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Antiphospholipid syndrome Symptoms
Views: 134 HealthSymptoms
Avoiding Blood Clots: Mayo Clinic Radio
 
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Dr. Rizwan Sohail offers tips for avoiding blood clots during travel
Views: 216 Mayo Clinic
What are the hematology effects antiphospholipid antibodies?
 
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Hematology: thrombophilia: Antiphospholipid antibodies: Antiphospholipid antibodies can be associated with an autoimmune hemolytic anemia and thrombocytopenia.
My experience having Factor Five Leiden, Pulminary embolisms, DVT's while being pregnant
 
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This is my story of developing dvts, and pulmonary embolisms while being pregnant . I experienced both dvts and pulmonary embolism in February 2011 while being 5 months pregnant , I ended up being hospitalized an being placed on lovenox an morphine , phyentynol , vicodin, and Tylenol with Codeine not all pain killers were administered at the same time they were switched back an forth as to not make me get addicted to any of them . I underwent a surgery to place an Optease retrievable filter (IVC) filter . I was later diagnosed with Hereditary Factor Five Leiden. I have not had a blood clot occurrence since an hoping it remains that way. I am not a doctor nor do I claim to be this video is not to influence any ones decision making . I'm just sharing my experience with blood clots .
Views: 730 Alyssa Nicole
Ancient Humans & Neanderthals Had Sex, Here's How It Changed Us Forever
 
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We've known for a while that ancient humans got busy with Neanderthals, but how exactly did that Neanderthal DNA affect modern humans? Ancient Humans Had A Crazy Sex Life - https://youtu.be/6fezsrCk5p0 Sign Up For The Seeker Newsletter Here - http://bit.ly/1UO1PxI Get 20% off http://www.domain.com domain names and web hosting when you use coupon code SEEKER at checkout! Read More: Our Brains Harbor "Residual Echo" of Neanderthal Genes https://www.nimh.nih.gov/news/science-news/2017/our-brains-harbor-residual-echo-of-neanderthal-genes.shtml "NIMH researchers have produced the first direct evidence that parts of our brains implicated in mental disorders may be shaped by a "residual echo" from our ancient past. The more a person's genome carries genetic vestiges of Neanderthals, the more certain parts of his or her brain and skull resemble those of humans' evolutionary cousins that went extinct 40,000 years ago." Neanderthal DNA Is Being Purged From Our Genomes https://www.seeker.com/neanderthal-dna-is-being-purged-from-our-genomes-2085233335.html "Another Neanderthal extinction is taking place now, and it's happening in our genomes, suggests new research that finds natural selection is slowly removing Neanderthal genetic variants from modern populations." Neanderthal-Human Sex Happened Earlier https://www.seeker.com/neanderthal-human-sex-happened-earlier-1770891192.html "Remains of a Neanderthal woman who lived around 100,000 years ago in the Altai Mountains of Siberia reveal that human and Neanderthals mated much earlier than previously thought. One or more of her relatives were actually humans, a new study shows." ____________________ Seeker inspires us to see the world through the lens of science and evokes a sense of curiosity, optimism and adventure. Visit the Seeker website http://www.seeker.com/shows/?utm_source=youtube&utm_medium=social&utm_campaign=seeker Subscribe now! http://www.youtube.com/subscription_center?add_user=dnewschannel Seeker on Twitter http://twitter.com/seeker Trace Dominguez on Twitter https://twitter.com/tracedominguez Seeker on Facebook https://www.facebook.com/SeekerMedia/ Seeker http://www.seeker.com/ This episode of Seeker was written and hosted by Trace Dominguez
Views: 604479 Seeker
IEF Project Spotlight: Therapeutic Plasma Levels of Aminocaproic Acid in Asian Elephants
 
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IEF Project Spotlight: Therapeutic Plasma Levels of Aminocaproic Acid in Asian Elephants (Final Report) The goal of this project was to determine if aminocaproic acid (EACA), a drug used to prevent uncontrolled bleeding, could be effective in Asian elephants. Elephants can experience uncontrolled bleeding from a variety of causes, including following an infection with the elephant endotheliotropic herpesvirus (EEHV). Due to their large size and limited blood banking resources, treatment options for an elephant with uncontrolled bleeding are severely limited. Aminocaproic acid is widely used in human and domestic animal medicine to stabilize blood clots and prevent hemorrhage, and may have applications to elephants as well. We succeeded in developing an in vitro model of hyperfibrinolysis (blood clot breakdown), mimicking a diseased state. We then succeeded in reversing this diseased state through addition of EACA. The elephant plasma required a lower concentration of EACA to inhibit fibrinolysis, compared to humans and dogs. Our results suggest that EACA may be effective in preventing bleeding in elephants in vivo, and may be a valuable treatment option for a variety of bleeding disorders, including animals with EEHV infection presenting with hemorrhage. For more information about this project, and other projects to protect elephants, visit www.elephantconservation.org
Views: 186 IntElephantFound
MS - Pathophysiology
 
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Views: 200 AliveMedCME
Factor V Leiden Awareness
 
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Blog http://chronicallygratefuldebla.com
Clinical and Laboratory Update in Thrombosis and Anticoagulation Conference
 
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Robert D. McBane, MD and Rajiv K. Pruthi, MBBS discuss the conference which will be held August 4-5, 2011 in Rochester, MN. An estimated 37 million patients are affected by thrombotic disorders each year. This number will continue to rise with the increase in our aging population. Clinical and Laboratory Update in Thrombosis and Anticoagulation will focus on clinically applicable and cutting-edge methods used in the evaluation and treatment of patients with arterial and venous thrombotic disorders.
Views: 454 Mayo Clinic
I Have Blood Clotting Disorders / INR Meter Testing
 
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I have 2 blood clotting disorders : Lupus Anticoagulant & Factor 5. I use an INR Meter to test my blood coagulation ( INR ) every Wednesday.
Views: 194 Big O Dill
Antiphospholipid Syndrome (clip)
 
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** To watch this video in full and claim a CPD credit please click here: http://www.rsmvideos.com/index.php/videoPlayer/?vid=319&class=videoThumbOdd Professor Anisur Rahman discusses Antiphospholipid Syndrome. He describes what the syndrome is and how to diagnose it. He also explains how to manage APS in pregnancy and suggests new tests and therapies for it. This Lecture was filmed at an event called 'Current theories and management of CTD' at the Royal Society of Medicine in London. Date of Lecture: 10th October 2012 The Royal Society of Medicine is an independent, apolitical organisation, founded over 200 years ago. We are one of the largest providers of continuing medical education in the UK.
Case of Severe Hypertension and Nephrotic Range Proteinuria
 
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Title: Case of Severe Hypertension and Nephrotic Range Proteinuria Description: Severe Hypertension and Nephrotic Range Proteinuria. Presented by Dr. Michael Bursztyn at the AHA Council on Hypertension | AHA Council on Kidney in Cardiovascular Disease | American Society of Hypertension Joint Scientific Sessions 2017 as part of a Clinical-Pathological conference session sponsored by Hypertension, an American Heart Association journal. Filmed 16 September 2017. The case follows a 37-year-old man referred to emergency for severe hypertension. His blood pressure (BP) in clinic was 186/125 mm Hg and he presented with headaches, nausea, dizziness and left flank pain.
Views: 223 AHAScienceNews
Focus on Recurrent Miscarriage Phenotypes
 
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Multiple, later-stage miscarriages may be a sign of treatable medical problems, such as diabetes or autoimmunity. Dr. David Barad discusses efforts to understand and treat recurrent miscarriages, for the medical journal fertility & sterility. Learn about recurrent miscarriages at: https://goo.gl/2hneGJ