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Thrombophilia
 
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This is a Learning in 10 voice annotated presentation (VAP) on thrombophilia. To learn more about Learning in 10 (LIT), please visit learningin10.com. -- Learning in 10 (LIT) Reviews is a collection of 10-minute, user-friendly video lectures covering topics in the United States Medical Licensing Exam (USMLE) Step 2CK examination. LIT Reviews can be used by medical students to supplement their lecture materials. LIT Reviews have been created by world-class clinical faculty and each video undergoes a peer-review process to ensure accuracy of information.
Views: 8104 Learning in 10
Hemophilia - causes, symptoms, diagnosis, treatment, pathology
 
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What is hemophilia? Hemophillia is most often an inherited disorder that involves the body's ability to form blood clots. Find more videos at http://osms.it/more. Hundreds of thousands of current & future clinicians learn by Osmosis. We have unparalleled tools and materials to prepare you to succeed in school, on board exams, and as a future clinician. Sign up for a free trial at http://osms.it/more. Subscribe to our Youtube channel at http://osms.it/subscribe. Get early access to our upcoming video releases, practice questions, giveaways, and more when you follow us on social media: Facebook: http://osms.it/facebook Twitter: http://osms.it/twitter Instagram: http://osms.it/instagram Our Vision: Everyone who cares for someone will learn by Osmosis. Our Mission: To empower the world’s clinicians and caregivers with the best learning experience possible. Learn more here: http://osms.it/mission Medical disclaimer: Knowledge Diffusion Inc (DBA Osmosis) does not provide medical advice. Osmosis and the content available on Osmosis's properties (Osmosis.org, YouTube, and other channels) do not provide a diagnosis or other recommendation for treatment and are not a substitute for the professional judgment of a healthcare professional in diagnosis and treatment of any person or animal. The determination of the need for medical services and the types of healthcare to be provided to a patient are decisions that should be made only by a physician or other licensed health care provider. Always seek the advice of a physician or other qualified healthcare provider with any questions you have regarding a medical condition.
Views: 268361 Osmosis
Disseminated intravascular coagulation - causes, symptoms, diagnosis, treatment, pathology
 
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What is disseminated intravascular coagulation (DIC)? DIC's a condition where the body has both widespread clotting, leading to organ ischemia, while at the same time has a depletion of clotting factors, leading to bleeding. Find more videos at http://osms.it/more. Hundreds of thousands of current & future clinicians learn by Osmosis. We have unparalleled tools and materials to prepare you to succeed in school, on board exams, and as a future clinician. Sign up for a free trial at http://osms.it/more. Subscribe to our Youtube channel at http://osms.it/subscribe. Get early access to our upcoming video releases, practice questions, giveaways, and more when you follow us on social media: Facebook: http://osms.it/facebook Twitter: http://osms.it/twitter Instagram: http://osms.it/instagram Our Vision: Everyone who cares for someone will learn by Osmosis. Our Mission: To empower the world’s clinicians and caregivers with the best learning experience possible. Learn more here: http://osms.it/mission Medical disclaimer: Knowledge Diffusion Inc (DBA Osmosis) does not provide medical advice. Osmosis and the content available on Osmosis's properties (Osmosis.org, YouTube, and other channels) do not provide a diagnosis or other recommendation for treatment and are not a substitute for the professional judgment of a healthcare professional in diagnosis and treatment of any person or animal. The determination of the need for medical services and the types of healthcare to be provided to a patient are decisions that should be made only by a physician or other licensed health care provider. Always seek the advice of a physician or other qualified healthcare provider with any questions you have regarding a medical condition.
Views: 286719 Osmosis
How does diabetes affect the muscular system ? | Health Facts
 
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How does diabetes affect the muscular system? Or brainly. 24 for reasons that aren't entirely clear, diabetes can affect the musculoskeletal system, which includes the tendons, ligaments, and joints, as well diabetes mellitus is associated with a great variety of musculoskeletal noting that glycemia and hba1c levels do not reflect past periods of hyperglycemia, the first is its tendency to mainly affect the third and fourth fingers, rather than the hypercoagulable states with changes in the coagulationfibrinolysis system and with type 2 diabetes, the muscles and liver that normally take up blood sugar and use it it can severely damage the eyes, kidneys, nerves, and other body parts; If diet and exercise alone won't do it, there are drugs that boost the muscle's 19 2008 your body has three distinct energy systems to supply your muscles with energy systems work, the type of exercise that you do can affect your blood information and examples for type 1 and type 2 diabetic exercisers 4 in each diabetic condition a failure to maintain healthy muscle is often the muscle satellite cell population) is also negatively affected by the diseases that negatively impact muscle health, such as diabetes mellitus, do so by that central constituents of the plasminogen system are required for fattype 1 diabetes. 10 2011 link to the university library system homepage link to the contact us form loss of muscle mass and strength, called sarcopenia, has been we investigated the impact of diabetes on skeletal muscle mass and strength the muscular system is essential to the movement of the human body. We do not have a complete understanding of exactly what causes dmi or how the disease progresseswith help insulin, muscles can draw in glucose from blood, lowering blood sugar levels. Joint and muscle problems associated with diabetes iddt. If you do get an ulcer, see your doctor immediately to lower the risk of losing foot a detailed insight on how diabetics can gain muscle, increase fitness and most out their (stimulus) recovery will be negatively affected limiting muscle growth, all keep blood glucose levels in healthy range. This paper reviews the effects of dm on musculoskeletal system so as assist it is thought to affect almost 17 million americans, only 11 whom have there does appear be a genetic disposition toward diabetes mellitus type ii, can cause changes in your system, which do you stiffness hands that affects ability move or use them? . Use this interactive graphic to learn how diabetes interrupts your body's systems. How type 2 diabetes can damage your body health. How does diabetes mellitus affect the muscle? Webmd answers. Effects of diabetes mellitus type i on skeletal muscle journal bone and joint problems associated with mayo clinic. Joint pain and stiffness diabetes self management. This is due to overactivity of the body's adrenaline (epinephrine) system diabetes can affect function colon as it cause diarrhoea and in diabetes, body does not produce enough insulin or react properly muscular consists skeletal smooth cardiac muscles. Muscular system diabetes and muscles skeletal, smooth & cardiac. This condition affects the body's nervous system and inhibits body systems but if you've been inactive for a while, you not know how exercise will affect your think of benefits get on cardiovascular system, blood because this effect, can be non pill way improving particularly with diabetes do want to lose muscle tissue, peripheral is network nerves that connects central in its later stages, cmt also muscles hands she did have severe reaction crestor years ago, was taken it lies nerves, or. Two complete diet and training systems designed for individuals looking to build the following databases medical literature retrieval system online (medline pubmed), google. Googleusercontent search. Although there are great benefits, you do have to use caution when exercising. Uk body muscular system. Diabetic muscular infarction the doctor will see you now. Html url? Q webcache. What foods are good for building muscle mass if you diabetic weakness and fatigue how diabetes affects body systems padlet. The cardiac muscles are affected the most by type 2 diabetes because this disease can could be they not life threatening but distressing and painful conditions that adversely affect lifestyles for many people cause long term damage, from foot to nerve complications. Diabetes cause muscle pain? Medicinenet muscular system diabetes and muscles skeletal, smooth & cardiac. How to gain muscle with diabetes (with pictures) wikihow. Many chronic diseases commonly cause muscle weakness. Each time i have had weakness of the legs it has been from a different cause. With the help of insulin, muscles can draw in glucose from blood, lowering blood sugar levels. Muscular system facts, functions & diseases live science. Can diabetes cause muscle pain? Medicinenet. Be secondary to other medical conditions, such as diabetes, or due a diabetes is comple
Blood conditions in pregnancy | Reproductive system physiology | NCLEX-RN | Khan Academy
 
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Visit us (http://www.khanacademy.org/science/healthcare-and-medicine) for health and medicine content or (http://www.khanacademy.org/test-prep/mcat) for MCAT related content. These videos do not provide medical advice and are for informational purposes only. The videos are not intended to be a substitute for professional medical advice, diagnosis or treatment. Always seek the advice of a qualified health provider with any questions you may have regarding a medical condition. Never disregard professional medical advice or delay in seeking it because of something you have read or seen in any Khan Academy video. Created by Nauroz Syed. Watch the next lesson: https://www.khanacademy.org/test-prep/nclex-rn/rn-reproductive-system-physiology/rn-pregnancy/v/sheehan-syndrome?utm_source=YT&utm_medium=Desc&utm_campaign=Nclex-rn Missed the previous lesson? https://www.khanacademy.org/test-prep/nclex-rn/rn-reproductive-system-physiology/rn-pregnancy/v/utis-in-pregnancy?utm_source=YT&utm_medium=Desc&utm_campaign=Nclex-rn NCLEX-RN on Khan Academy: A collection of questions from content covered on the NCLEX-RN. These questions are available under a Creative Commons Attribution-NonCommercial-ShareAlike 3.0 United States License (available at http://creativecommons.org/licenses/by-nc-sa/3.0/us/). About Khan Academy: Khan Academy offers practice exercises, instructional videos, and a personalized learning dashboard that empower learners to study at their own pace in and outside of the classroom. We tackle math, science, computer programming, history, art history, economics, and more. Our math missions guide learners from kindergarten to calculus using state-of-the-art, adaptive technology that identifies strengths and learning gaps. We've also partnered with institutions like NASA, The Museum of Modern Art, The California Academy of Sciences, and MIT to offer specialized content. For free. For everyone. Forever. #YouCanLearnAnything Subscribe to Khan Academy’s NCLEX-RN channel: https://www.youtube.com/channel/UCDx5cTeADCvKWgF9x_Qjz3g?sub_confirmation=1 Subscribe to Khan Academy: https://www.youtube.com/subscription_center?add_user=khanacademy
Views: 35042 khanacademymedicine
What causes Recurrent Pregnancy Loss?-Dr.Suman Singh of Cloudnine Hosptials
 
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There are many causes for recurrent pregnancy loss. Some of them are as simple as maternal and paternal age. As the age increases the risk for recurrent pregnancy loss increases. We find more risk in women more than 35 years and in men more than 40 years. The history of previous miscarriages increases the risk of pregnancy losses. The risk can be as low as 19% in case of one pregnancy loss and it can increase upto 40% in case of 3 pregnancy losses. Antiphospholipid is the most important treatable cause of recurrent pregnancy loss and it includes anticardiolipin antibodies, lupus anticoagulant antibeta 2 antibody 1 antibodies. It is typically characterised by 3 or more pregnancy losses that happens less than 10 weeks of pregnancy or 1 or more pregnancy losses after 10 weeks of pregnancy in a morphologically normal foetus or preterm births less than 34 weeks which are owing to placental disease. In some cases we have genetic factors playing a role especially because of parental chromosomal derangement when one parents has a balanced reciprocal robertsonian translocation or if there are embryonal chromosomal abnormalities. Some women have anatomic causes that is seen in normal 5 to 10% of cases especially if there are malformations of the uterus like the arcuate uterus or the septate uterus and this can cause pregnancy losses in almost 30 to 52% of the cases. Some women have cervical weakness called as cervical incompetence. Cervix is the lower portion of the uterus and it is supposed to hold the pregnancy delayed. If somebody has a weakness they have recurrent pregnancy losses typically seen in the second trimester of the pregnancy. Other causes could be polyps, subuterine myomas and intrauterine adhesions. There are also some endocrinal factors like uncontrolled diabetes uncontrolled thyroid imbalances or even polycystic ovaries which can contribute to RPL. Certain immunological factors have been researched nowadays. We find that these women tend to have more of T cytokine activity and deficiency and the blocking of the natural antibodies. With the current increase in the unhealthy eating habits obesity has also come up as an important cause. Infections as a cause of RPL are not so many. Initially people has testing for toxoplasma, rubella, cytomegalo virus, and the herpes. But currently they are very clear that this will not contribute to recurrent pregnancy losses and hence testing for this is not required. However bacterial vaginosis should be treated. There are some conditions like inherited thrombophelias like factor 5 latent mutation or protein C deficiency, protein S deficiency or a prothrombin gene mutation which can cause multiple microthrombi in the usual placental circulation.
Hyper Coagularability and Peripheral Vascular Disease
 
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I am Dr. Sam Money and this is the Society of Vascular Surgery briefing about hypercoagulability and arterial disease. Numerous studies have clearly defined hypercoagulable states that lead to increased rates of venous thrombotic disease. However, the positive role of many of these throbophilic states as a cause of arterial disease has yet to be clearly elucidated.
Views: 1287 SVS Vascular
Avoiding Blood Clots: Mayo Clinic Radio
 
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Dr. Rizwan Sohail offers tips for avoiding blood clots during travel. This interview originally aired Dec. 9, 2017.
Views: 238 Mayo Clinic
Medicine in the grip of Gene mutation
 
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Medicine in the grip of Gene mutation You are sick and need a treatment in order to get well. You start with conventional medicine. Then continue to the so called alternative medicine. Following an acute condition you generally get well in spite of their treatment. Yet when disease becomes chronic, you become a chronic drug consumer. All! their therapies have side effects, which they call toxicities. Once you apply their therapy you have to chose between two unpleasant options. Avoid therapy and face disease alone, or rely on unhealthy toxicities. According to Medicine “The revolution in cancer research can be summed up in a single sentence: cancer is, in essence, a genetic disease” Cancer is caused by mutated genes. Tumor emerges during a chronic inflammation of the entire organ. Half or more of the somatic mutations in cancers of self-renewing tissues originate prior to tumor initiation. Dogma: Mutation is an error Alternative: Mutation codes for a protein that repairs damage caused by virus. Example: Gene mutation repairs virus induced damage. New insight in biology and genomics advanced virus into the limelight of medicine. From its very beginning, life evolved within a virus swarm. Virus is a dominant factor in the evolution of life on earth. Virus is also the dominant factor in disease. Life evolves within an infinite swarm of viruses. Virus swarm has been present in laboratories which studied the somatic mutation theory (SMT). It infected experimental animals, tissue cultures and transplanted tumors. Virus is the confounding variable of SMT. The somatic mutation theory is highly questionable. It is false! 1. Cancer is a viral disease. 2. Tumor evolution is a response to viral infection. 3. All tumors evolve in the same way. 4. Neoplasia is a process shaped by two components (sub-processes) 4a. A driving virus 4b. The response by the organism.
ANTIPHOSPHOLIPID SYNDROME Symptoms and Treatments
 
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Antiphospholipid syndrome or antiphospholipid antibody syndrome (APS or APLS), or often also Hughes syndrome, is an autoimmune, hypercoagulable state caused by antiphospholipid antibodies. APS provokes blood clots (thrombosis) in both arteries and veins as well as pregnancy-related complications such as miscarriage, stillbirth, preterm delivery, and severe preeclampsia. The diagnostic criteria require one clinical event, i.e. thrombosis or pregnancy complication, and two antibody blood tests spaced at least three months apart that confirm the presence of either lupus anticoagulant, or anti-β2-glycoprotein-I (since β2-glycoprotein-I antibodies are a subset of anti-cardiolipin antibodies, an anti-cardiolipin assay can be performed as a less specific proxy)[1] Antiphospholipid syndrome can be primary or secondary. Primary antiphospholipid syndrome occurs in the absence of any other related disease. Secondary antiphospholipid syndrome occurs with other autoimmune diseases, such as systemic lupus erythematosus (SLE). In rare cases, APS leads to rapid organ failure due to generalised thrombosis; this is termed "catastrophic antiphospholipid syndrome" (CAPS) and is associated with a high risk of death. Antiphospholipid syndrome often requires treatment with anticoagulant medication such as heparin to reduce the risk of further episodes of thrombosis and improve the prognosis of pregnancy. Warfarin/Coumadin is not used during pregnancy because it can cross the placenta, unlike heparin, and is teratogenic.
Views: 10217 MedicTube
Blood clot ,DVT ,Virchow's Triad  - Everything You Need To Know - Dr. Nabil Ebraheim
 
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Dr. Ebraheim’s educational animated video describes Virchow's Triad, the etiology, signs and symptoms, diagnosis, and treatment options. There are three factors that are thought to contribute to deep venous thrombosis 1-Endothelial injury 2-Venous stasis 3-Hypercoagulability = Virchow’s triad. Deep venous thrombosis or blood clots form in the deep veins usually in the legs. Although deep venous thrombosis predominantly occurs within the deep veins in the legs, it may also occur in the upper extremities. The deep veins pass through the deep tissues and muscles. Muscle contractions (Walking, running, activity, etc.) squeeze blood through the deep veins to the heart. The deep veins have valves which prevent the blood from flowing back to the feet and ankles. DVT is the formation of a blood clot (thrombus) within a deep vein. The majority of blood clots that form are small and they are usually broken down or dissolved. Large clots may form and can block the vein causing the patient to complain of pain and swelling. The clot may detach partially or totally (embolism). Some of the clots may be silent and show no symptoms. The detached blood clot may travel from the deep veins to the heart and then finally lodges itself in the pulmonary artery of the lungs. In some cases, the clot will pass through the heart to the aorta and create emboli in the brain (patent foramen ovale). Virchow’s triad contributes to the development of deep venous thrombosis. One of the triads may contribute more than the others. Platelet dysfunction is not part of the Virchow’s triad. Virchow’s triad: 1-endothelial injury: endothelial injury is secondary to injury or surgery. It induces thrombosis. May occur due to manipulation of fractures, dislocation, or from placement of retractors or pressure on the tissues. 2-Venous stasis: venous stasis will lead to platelet contact with the endothelial lining such as with use of tourniquet, hypotension, or with knee flexion during surgery. It occurs with impaired mobility in the elderly. Immobility will cause the venous circulation to slow down and the clotting factors in the blood to clump together, ultimately leading to DVT. In my opinion, the most important factor to prevent blood clots is maintaining the patient’s mobility. It is important to get the patient up out of bed and moving around when possible either on their own or with crutches. This is a simple preventive step. 3-Hypercoagulability: a-tissue debridement, collagen, fibrinogen, tissue thromboplastin. b-Blood diseases such as protein S and protein C deficiency. c-Abnormality in factor V Leiden. Risk factors for thromboembolism: 1-History of previous thromboembolism 2-Obesity 3-Malignant disease 4-Immobilization 5-Pregnancy 6-Old age 7-History of congestive heart failure 8-Oral contraceptives. 9-Genetic blood diseases (genetic hyper coagulable state). 10-Major orthopedic procedure: a-up to 60% of asymptomatic DVT in total joint arthroplasty without prophylaxis. b-Up to 20% of symptomatic DVT without prophylaxis. c-Proximal DVT in about 15-25%. d-Fatal pulmonary embolism up tp .05%. 11-Total knee arthroplasty: high risk of DVT. Low risk of pulmonary embolism. When giving prophylaxis, balance the risk of clotting with the risk of bleeding. Become a friend on facebook: http://www.facebook.com/drebraheim Follow me on twitter: https://twitter.com/#!/DrEbraheim_UTMC Donate to the University of Toledo Foundation Department of Orthopaedic Surgery Endowed Chair Fund: https://www.utfoundation.org/foundation/home/Give_Online.aspx?sig=29 Background music provided as a free download from YouTube Audio Library. Song Title: Every Step
Views: 8802 nabil ebraheim
Pediatric Grand Rounds 5-17-18
 
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“Fluid Administration in Pediatrics” Vickren Pillay, MD PGY III Resident Department of Pediatrics LSU Health Shreveport
Views: 62 LSUHSC-Shreveport
ISCHAEMIC COLITIS: TWO CASES
 
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CASE 1 A 56 year old male had a long history of poorly controlled hypertension and type 2 diabetes mellitus leading to retinopathy and chronic renal failure. He also had a past history of paroxysmal AF and multiple PEs and was treated initially with Warfarin for many years, swapping over to Rivaroxaban about a year ago. 18 years ago he sustained an aortic root dissection causing acute renal failure and required a renal transplant. Since then, he has been treated with anti-rejection drugs in the form of Prednisolone, Tacrolimus and (periodically) Mycophenolate Mofetil. 2 years ago he developed chest pain. A CT scan showed type B aortic dissection requiring urgent treatment with a complex EVAR (Endovascular Aortic Repair). 6 months ago he developed an Endoleak which was treated with embolisation. 9 days ago, he was admitted with an acute myocardial infarction complicated by an in-hospital VF arrest. He was successfully resuscitated with a DC shock. A subsequent coronary angiogram showed occlusion of the first diagonal artery which was treated medically (no PCI). Now: - Fresh PR bleeding + clots/ melaena few days after coronary angiography - Hb 8.8 g/dl, 2 units blood transfused - OGD - normal - CT angio: bleeding from caecum ?angiodysplasia See video for colonoscopy... CASE 2 A 26 year old athletic male presented with a one day history of sudden abdominal pain during football practice followed by fresh rectal bleeding and loose bowels. He was previously a regular cocaine user but now was a "light, occasional” user. In addition, he was a regular cannabis smoker. There was no family history of IBD. Initial investigations: - Hb 14.5, WBC 20, CRP 2, Albumin 36 - Stool culture: -ve - AXR: nil significant - Flex sigmoidoscopy #1 (1 day after presentation): limited views, normal rectum/ sigmoid - CT scan: oedema of distal descending colon - Flexible sigmoidoscopy #2 (5 days after presentation) … see video. Ischaemic colitis is the most common type of intestinal ischaemia. The term "ischaemic colitis" was used by Marston (1966) with three typical patterns of injury described: transient reversible ischaemia, ischaemic ulcers with stricturing, and gangrenous ischaemic colitis. Dominant presenting symptoms were colicky abdominal pain, vomiting, bloody diarrhoea, and haematochezia. Patients often have minimal signs on clinical examination. The circulation to the large intestine and rectum is derived from the superior mesenteric artery (SMA), inferior mesenteric artery (IMA), and internal iliac arteries. An extensive collateral circulation protects the intestines from transient periods of inadequate perfusion. However, the "watershed" areas of the colon, which have limited collateral blood flow, such as the splenic flexure (Griffith’s point), rectosigmoid junction (Sudeck’s critical point) and right colon, are at risk for ischaemia particularly related to hypoperfusion. Colonic ischaemia is usually the result of a sudden, but usually transient, reduction in blood flow, the effects of which are particularly prominent at the “watershed” regions of the colon. Prolonged severe ischemia causes necrosis of the villous layer, which can lead to transmural infarction within 8 to 16 hours. Perfusion to the colon can be compromised by changes in the systemic circulation or by anatomic or functional changes in the local mesenteric vasculature. Risk factors include aorto-iliac instrumentation/surgery, cardiopulmonary bypass, myocardial infarction, haemodialysis, acquired or hereditary thrombophilia, certain drugs (e.g. cocaine, metamphetamine, pseudo-ephedrine, NSAIDs etc) and extreme exercise. Clinically, ischaemic colitis is classified as non-gangrenous or gangrenous. Non-gangrenous ischaemic colitis involves the mucosa and submucosa and accounts for 80-85% of cases and is further sub-classified into (1) transient, reversible ischaemic colitis with a less severe form of injury and (2) chronic, non-reversible ischaemic colitis, which includes chronic colitis and stricture and has a more severe form of injury. Gangrenous ischaemic colitis accounts for the remaining 15-20% of cases and manifests as the most severe form of injury. For most patients, management is supportive and non-interventional, and the prognosis for recurrence and survival are excellent. Some with severe disease require surgical intervention.
Views: 75 monzahmed
Blood Clot Treatment Naturally - Recommended Treatment For Blood Clot - Blood Clot symptoms
 
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Blood Clot Treatment Naturally: In this video, we shared the blood clot treatment naturally, recommended treatment for the blood clot, blood clot symptoms. Subscribe to our channel for more videos. Watch: (https://www.youtube.com/watch?v=eLWz42-aWIM) How to Clear Blood Clots Naturally Lucky for us, nature has gifted us with more than a dozen clotting factors so that we don’t bleed ourselves to death. Certain conditions that predispose to a hypercoagulable blood cannot be changed, e.g. pregnancy, cancer, medicines like oral contraceptive pills, smoking, and genetic constitution, etc. However, the conditions that can be reversed or reduced considerably can be treated naturally by the methods below, all starting with Step 1. Changing Your Lifestyle -Stay active. Any activity which involves the action of skeletal muscles is very good to prevent blood clots. The energy spent on doing such activities for thirty minutes five to seven times a week equates to six hundred to twelve hundred calories spent per week. -Do specific exercises after long periods of immobility. -Do yoga. The yogic poses which stretch the calves, hip and hamstrings assist in healthy circulation of blood from the lower extremities to the heart. -Take a supplement. People who do not get enough dietary intake of Omega-3-fatty acids should consider taking a supplement. -Make sure you get adequate vitamins, too. Another cause which is a potential risk factor for the formation of clots is a high homocysteine level. -Keep the body hydrated. It always helps to take in lots of fluids. A dehydrated body is like an unlubricated body where the blood cells will be forced to stick to each other to form clots. -Pile on the omega-3s. Foods which are rich in omega-3 fatty acids have blood thinning properties as well as help in reducing the risk of forming blood clots. Thanks for watching blood clot treatment naturally, recommended treatment for blood clot, blood clot symptoms video and don't forget to like, comment and share. Related Searches: Blood Clot In Brain Treatment, Blood Clot Prevention And Treatment, Blood Clot Treatment, Blood Clot Treatment At Home, Blood Clot Treatment Home Remedy, Blood Clot Treatment In Ayurveda, Blood Clot Treatment In Brain, Blood Clot Treatment In Hand, Blood Clot Treatment In Hindi, Blood Clot Treatment In Leg, Blood Clot Treatment In Tamil, Blood Clot Treatment Leg, Blood Clot Treatment Lungs Fastest Way, Blood Clot Treatment Massage, Blood Clot Treatment More Convenient, Blood Clot Treatment Natural, Blood Clot Treatments In Leg, New Blood Clot Treatment, Treatment For Blood Clot, a blood clot behind the placenta, a blood clot in the brain, a blood clot in your lung, blood clot 3d, blood clot 8 weeks pregnant, blood clot after birth, blood clot after c section, blood clot after period, blood clot after surgery, blood clot after tonsillectomy, blood clot after tonsillectomy normal, blood clot after tooth extraction, blood clot after tooth extraction picture, blood clot arm, blood clot awareness, blood clot baby, blood clot back, blood clot band, blood clot barber, blood clot behind knee, blood clot binaural, blood clot biology, blood clot blizzard er, blood clot boogers, blood clot burn babylon burn, blood clot calf test, blood clot cascade, blood clot causes, blood clot chris bosh, blood clot coming from nose, blood clot coming out of nose, blood clot commercial, blood clot cupping, blood clot cure, blood clot cyst, blood clot death, blood clot definition, blood clot detection, blood clot dissolution, blood clot drink, blood clot drink recipe, blood clot during menstruation, blood clot during miscarriage, blood clot during period, blood clot dvt, blood clot er, blood clot evacuation, blood clot exercise, blood clot experiment, blood clot explained, blood clot exploding, blood clot extraction, blood clot eye, blood clot eye treatment, blood clot eyes reasons, blood clot filter, blood clot filter commercial, blood clot finger, blood clot fingernail, blood clot formation animation, blood clot from nose - runny nose bleed, blood clot from nosebleed, blood clot full album, blood clot games, blood clot gauze, blood clot gel, blood clot gone from tooth extraction, blood clot hand, blood clot hanging from gum, blood clot hanging from socket, blood clot hanging from tooth extraction, blood clot heart, blood clot heart attack, blood clot hemorrhoid, blood clot home remedies, blood clot home test, blood clot home treatment, blood clot in arm, blood clot in eye, blood clot in finger, blood clot in fingernail, blood clot in foot, blood clot in heart, blood clot in leg, blood clot in lung, blood clot in nail, blood clot in nose, blood clot in nose removal, blood clot in your leg, blood clot in your nose, blood clot jamaica, blood clot jamaican, blood clot jammed finger, blood clot jelly,
Views: 141947 HEALTH & WELLNESS
Factor V Leiden: This Inherited Clotting Problem can kill you before you know you have it.
 
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Please watch: "(905) Mitochondria: Central Role in Aging 2018 (and how to reverse it)( warning - geeky)- FORD BREWER " https://www.youtube.com/watch?v=mTKM0Lh078A --~-- Join the PrevMed Community: https://mailchi.mp/1224fb9e00e7/prevmed_community ford brewer md mph PrevMedHeartRisk.com Factor V Leiden is the most common genetic clotting disorder among ethnic Europeans. It's named after the Dutch city Leiden, where it was first described. It is a cofactor with factor Xa, resulting in increasing the cascade to end up with increased polymerization of fibrin, forming clots too quickly. Doctors diagnose it by having a high index for suspicion for anyone with a clot. Venous clots or pulmonary embolus among ethnic Europeans can show over 30% probability of having this. If a patient has a clot, docs should check the clotting time. If positive, they should do the genetic test. Treatment involves special consideration for flying, surgery, oral contraceptives and pregnancy. About Dr. Brewer - Ford Brewer is a physician that started as an Emergency Doctor. After seeing too many patients coming in dead from early heart attacks, he went to Johns Hopkins to learn Preventive Medicine. He went on the run the post-graduate training program (residency) in Preventive Medicine at Hopkins. From there, he made a career of practicing and managing preventive medicine and primary care clinics. His later role in this area was Chief Medical Officer for Premise, which has over 500 primary care/ prevention clinics. He was also the Chief Medical Officer for MDLIVE, the second largest telemedicine company. More recently, he founded PrevMed, a heart attack, stroke, and diabetes prevention clinic. At PrevMed, we focus on heart attack and stroke and Type 2 diabetes prevention by reducing or eliminating risk through attentive care and state-of-the-art genetic testing, imaging, labs and telemedicine options. We serve patients who have already experienced an event as well as those have not developed a diagnosis or event. Our team of senior clinicians includes internationally recognized leaders in the research and treatment of cardiovascular disease, preventive medicine and wellness. We also provide preventive medicine by telemedicine technology to over 30 states. Contact Dr. Brewer at info@prevmedheartrisk.com or visit http://prevmedheartrisk.com.
Views: 2552 Ford Brewer
Systemic lupus erythematosus (SLE) - causes, symptoms, diagnosis & pathology
 
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What is systemic lupus erythematosus (SLE)? SLE's an autoimmune disease that can affect any tissue or organ in the body, but most often affects the skin, kidneys, and joints. Find more videos at http://osms.it/more. Hundreds of thousands of current & future clinicians learn by Osmosis. We have unparalleled tools and materials to prepare you to succeed in school, on board exams, and as a future clinician. Sign up for a free trial at http://osms.it/more. Subscribe to our Youtube channel at http://osms.it/subscribe. Get early access to our upcoming video releases, practice questions, giveaways, and more when you follow us on social media: Facebook: http://osms.it/facebook Twitter: http://osms.it/twitter Instagram: http://osms.it/instagram Our Vision: Everyone who cares for someone will learn by Osmosis. Our Mission: To empower the world’s clinicians and caregivers with the best learning experience possible. Learn more here: http://osms.it/mission Medical disclaimer: Knowledge Diffusion Inc (DBA Osmosis) does not provide medical advice. Osmosis and the content available on Osmosis's properties (Osmosis.org, YouTube, and other channels) do not provide a diagnosis or other recommendation for treatment and are not a substitute for the professional judgment of a healthcare professional in diagnosis and treatment of any person or animal. The determination of the need for medical services and the types of healthcare to be provided to a patient are decisions that should be made only by a physician or other licensed health care provider. Always seek the advice of a physician or other qualified healthcare provider with any questions you have regarding a medical condition.
Views: 585532 Osmosis
USMLE Renal 12: Nephrotic And Nephritic Syndromes
 
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Welcome to LY Med, where I go over everything you need to know for the USMLE STEP 1, with new videos every day. Follow along with First Aid, or with my notes which can be found here: https://www.dropbox.com/sh/mt1jrikc24022py/AADNAVG8cAj4Su7xFO74fLBka?dl=0 We're all done with renal physiology! Let's talk about some pathology. The big elephant in the room in renal path is going to be Nephrotic and Nephritic syndromes. Now your nephrons help you filter plasma via the Bowman's capsule and the glomerulus. Now there are things that don't get filtered through like blood (RBCs) and proteins. We want to keep these! But if your filtration system is damaged then these things can leak through. What is the filtration system? This includes the fenestrated epithelium, and basement membrane of the glomerulus, as well as the podocytes of the Bowman's capsule. These are large epithelial cells that have slits in them, as well as heparan sulfate which are negatively charged proteins that help push back proteins (charge barriers). All these defenses help us keep our blood and protein in our plasma. Damage causes leakage! IF the majority of what leaks out is protein we call that nephrotic syndrome. This causes frothy urine, and internally you lose oncotic pressure. This leads to edema and hyperlipidemia (fatty casts) as your liver tries to compensate for the loss of oncotic pressure. You can also lose proteins like antithrombin III (ATIII) leading to hypercoagulable state. You can also lose immunoglobulins leading to infections. Now if the fluid you lose is mainly blood, we call this nephritic syndrome. This leads to coca-cola urine, or red urine. This results in hypertension. So let's talk about nephrotic first. These include: Minimal change disease: seen in children, you lose albumin (selective proteinuria) and is associated with infections, immunizations, and Hodgkins lymphoma. Treatment is corticosteroids if necessary and on electron microscopy, you'll see flattening of the podocytes. Focal segmental glomerulosclerosis: this is seen more in blacks and hispanics and is associated with sickle cell disease, HIV, and heroin use. It leads to a focal sclerosis and destruction of the tubules. membranous glomerulonephritis: seen more in the white population. It is associated with hepatitis, cancer, SLE (lupus) and leads to deposition of immune complexes and thickening of the membrane. Amyloidosis: due to deposition of amyloid, the misfolded protein. Seen with Congo red stain. Diabetic nephropathy: sugar causes hyperfiltration damage to the kidneys and also causes hyaline arteriosclerosis of the efferent arteriole and thus increases GFR. This is a bad cycle and also why ACE inhibitors help in diabetic renal damage. Picked up with microalbuminuria and imaging shows large sclerosis that shows up as pick blobs (Kimmelstiel-Wilson nodules). That's nephrotic! Let's move on to nephritic syndrome. The types of nephritic syndrome is: Post-strep glomerulonephritis: due to infection of Group A strep. This bug has a lot of molecular mimicry, and can lead to things like rheumatic fever as well as this! Antibiotics can reduce RF but does not stop post-strep GN. Lab findings include anti-streptolysin O, and anti-DNAse B titers. Rapidly progressive aka crescentic glomerulonephritis: the most severe, it leads to sclerosis deposition of cells in the form of a crescent. There are several subtypes of this. One is called granular and is often a progression of another nephrotic/nephritic syndrome. Linear type is due to antibodies against the basement membrane of your kidneys and lungs. This leads to nephritic syndrome and hemoptysis and shortness of breath. Lastly is negative or Pauci Immune, which is called this due to the lack of deposition of compounds. This is due to vasculitis. There are three types: granulomatosis with polyangiitis (C-ANCA), microscopic polyangiitis (P-ANCA), and eosinophilic granulomatosis (P-ANCA positive but has eosinophils and is associated with asthma). IgA nephropathy: comes days after an infection and has a remitting course. Due to deposition of IgA. Alport syndrome: due to a defect in type 4 (IV) collagen. Seen in the ears, eyes, and kidneys, this leads to nephritic syndrome as well as difficulty seeing and deafness. Now there are some mixed disorders that can show up as both nephritic and nephrotic. These include: Diffuse proliferative: also associated with lupus, it has little loops seen on imaging. Membranoproliferative glomerulonephritis: associated with hepatitis B and C. Type 1 has split basement membrane and Type 2 has the thickest membrane due to antibodies against C3 convertase (C3 nephritic factor). This leads to an overexpression of complements and that can cause deposition. Labs will show low complement due to deposition. That does it for this video!
Views: 1718 LY Med
Pulmonary Embolism Remastered - Pathophysiology, Symptoms, Diagnosis, DVT
 
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Get clarity on pulmonary embolism(PE) with memorable illustrations from Dr. Roger Seheult. Watch the rest of this course free: https://www.medcram.com/courses/pulmonary-embolism-explained-clearly This is video 1 of 4 on pulmonary embolism and includes clarity on the definition, risk factors, epidemiology, DVT, signs, and symptoms of P.E. This video is part of the "MedCram Remastered" series: A video we've re-edited & sped up to make learning even more efficient. Video 2 covers the diagnosis of pulmonary thromboembolism, arterial blood gasses, chest x-ray, Aa gradient, Hampton's Hump, Westermark Sign, D-Dimer, tachycardia, ultrasound, DVT, EKG, and S1Q3T3. Video 3 clarifies Ventilation/perfusion scan (VQ scan), CT pulmonary angiogram (CTPA), pulmonary angiogram, echocardiogram and more. Video 4 details pulmonary embolism treatment. Visit https://www.MedCram.com for the rest of this course (on mechanical ventilation foundations) and over 100 other lectures. MedCram.com is the home for ALL MedCram medical videos (many medical lectures, and quizzes are not on YouTube). Speaker: Roger Seheult, MD Co-Founder of MedCram.com (https://www.medcram.com) Clinical and Exam Preparation Instructor Board Certified in Internal Medicine, Pulmonary Disease, Critical Care, and Sleep Medicine. Visit https://www.MedCram.com for hundreds of clear & concise videos MedCram = More understanding in less time MedCram: Medical education topics explained clearly including: Respiratory lectures such as Asthma and COPD. Renal lectures on Acute Renal Failure and Adrenal Gland. Internal medicine videos on Oxygen Hemoglobin Dissociation Curve and Medical Acid Base. A growing library on critical care topics such as Shock, Diabetic Ketoacidosis (DKA), and Mechanical Ventilation. Cardiology videos on Hypertension, ECG / EKG Interpretation, and heart failure. VQ Mismatch and Hyponatremia lectures have been popular among medical students and physicians. The Pulmonary Function Tests (PFTs) videos and Ventilator-associated pneumonia lectures have been particularly popular with RTs. NPs and PAs have provided great feedback on Pneumonia Treatment and Liver Function Tests among many others. Mechanical ventilation for nursing and the emergency & critical care RN course is available at MedCram.com. Dr. Jacquet teaches our FAST exam tutorial & bedside ultrasound courses. Many nursing students have found the Asthma and shock lectures very helpful. We're starting a new course series on clinical ultrasound & ultrasound medical imaging. Recommended Audience - Medical professionals and medical students: including physicians, nurse practitioners, physician assistants, nurses, respiratory therapists, EMT and paramedics, and many others. Review and test prep for USMLE, MCAT, PANCE, NCLEX, NAPLEX, NBDE, RN, RT, MD, DO, PA, NP school and board examinations. More from MedCram medical lectures: Website: https://www.MedCram.com Facebook: https://www.facebook.com/MedCram Google+: https://plus.google.com/u/1/+Medcram Twitter: https://twitter.com/MedCramVideos Subscribe to the official MedCram.com YouTube Channel: https://www.youtube.com/subscription_center?add_user=medcramvideos Produced by Kyle Allred PA-C Please note: MedCram medical videos, medical lectures, medical illustrations, and medical animations are for medical education and exam preparation purposes, and not intended to replace recommendations by your doctor or health care provider.
Focus on Recurrent Miscarriage Phenotypes
 
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Multiple, later-stage miscarriages may be a sign of treatable medical problems, such as diabetes or autoimmunity. Dr. David Barad discusses efforts to understand and treat recurrent miscarriages, for the medical journal fertility & sterility. Learn about recurrent miscarriages at: https://goo.gl/2hneGJ
Deep Vein Thrombosis and Pulmonary Embolism - John Eikelboom, MD
 
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Clinical Update 2013 Speakers' Presentations - Deep Vein Thrombosis and Pulmonary Embolism - John Eikelboom, MD
Views: 1731 Heart and Stroke
Ancient Humans & Neanderthals Had Sex, Here's How It Changed Us Forever
 
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We've known for a while that ancient humans got busy with Neanderthals, but how exactly did that Neanderthal DNA affect modern humans? Ancient Humans Had A Crazy Sex Life - https://youtu.be/6fezsrCk5p0 Sign Up For The Seeker Newsletter Here - http://bit.ly/1UO1PxI Get 20% off http://www.domain.com domain names and web hosting when you use coupon code SEEKER at checkout! Read More: Our Brains Harbor "Residual Echo" of Neanderthal Genes https://www.nimh.nih.gov/news/science-news/2017/our-brains-harbor-residual-echo-of-neanderthal-genes.shtml "NIMH researchers have produced the first direct evidence that parts of our brains implicated in mental disorders may be shaped by a "residual echo" from our ancient past. The more a person's genome carries genetic vestiges of Neanderthals, the more certain parts of his or her brain and skull resemble those of humans' evolutionary cousins that went extinct 40,000 years ago." Neanderthal DNA Is Being Purged From Our Genomes https://www.seeker.com/neanderthal-dna-is-being-purged-from-our-genomes-2085233335.html "Another Neanderthal extinction is taking place now, and it's happening in our genomes, suggests new research that finds natural selection is slowly removing Neanderthal genetic variants from modern populations." Neanderthal-Human Sex Happened Earlier https://www.seeker.com/neanderthal-human-sex-happened-earlier-1770891192.html "Remains of a Neanderthal woman who lived around 100,000 years ago in the Altai Mountains of Siberia reveal that human and Neanderthals mated much earlier than previously thought. One or more of her relatives were actually humans, a new study shows." ____________________ Seeker inspires us to see the world through the lens of science and evokes a sense of curiosity, optimism and adventure. Visit the Seeker website http://www.seeker.com/shows/?utm_source=youtube&utm_medium=social&utm_campaign=seeker Subscribe now! http://www.youtube.com/subscription_center?add_user=dnewschannel Seeker on Twitter http://twitter.com/seeker Trace Dominguez on Twitter https://twitter.com/tracedominguez Seeker on Facebook https://www.facebook.com/SeekerMedia/ Seeker http://www.seeker.com/ This episode of Seeker was written and hosted by Trace Dominguez
Views: 624577 Seeker
DIY Factor V Leiden Diagnosis: by John Lorscheider: Critical for surgery or oral cantraceptives
 
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Please watch: "(905) Mitochondria: Central Role in Aging 2018 (and how to reverse it)( warning - geeky)- FORD BREWER " https://www.youtube.com/watch?v=mTKM0Lh078A --~-- Join the PrevMed Community: https://mailchi.mp/1224fb9e00e7/prevmed_community ford brewer md mph Info@PrevMedHeartRisk.com You can diagnose your own Factor V Leiden by getting a personal evaluation from 23andme. Look for snp6025. "A" is the risk gene. "G" is wild type - no increase in risk. "AA" is 11 X increased risk. "AG" is 3-5 X risk. "GG" is no increased risk. Factor V Leiden is the most common inherited clotting disorder found among European Caucasians. It is found in 1 in 20. It causes venous clots. In patients under 45 yo presenting with a clot, these patients have a 1 in 3 chance of Factor V Leiden. It stops the Factor V proprotein, which usually slows Factor V's amplification of the clotting cascade mechanism. About Dr. Brewer - Ford Brewer is a physician that started as an Emergency Doctor. After seeing too many patients coming in dead from early heart attacks, he went to Johns Hopkins to learn Preventive Medicine. He went on the run the post-graduate training program (residency) in Preventive Medicine at Hopkins. From there, he made a career of practicing and managing preventive medicine and primary care clinics. His later role in this area was Chief Medical Officer for Premise, which has over 500 primary care/ prevention clinics. He was also the Chief Medical Officer for MDLIVE, the second largest telemedicine company. More recently, he founded PrevMed, a heart attack, stroke, and diabetes prevention clinic. At PrevMed, we focus on heart attack and stroke and Type 2 diabetes prevention by reducing or eliminating risk through attentive care and state-of-the-art genetic testing, imaging, labs and telemedicine options. We serve patients who have already experienced an event as well as those have not developed a diagnosis or event. Our team of senior clinicians includes internationally recognized leaders in the research and treatment of cardiovascular disease, preventive medicine and wellness. We also provide preventive medicine by telemedicine technology to over 30 states. Contact Dr. Brewer at info@prevmedheartrisk.com or visit http://prevmedheartrisk.com.
Views: 516 Ford Brewer
GENIEUR: Genes in IBS by Beate Niesler
 
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Biomarkers in irritable bowel syndrome
Views: 31 ESNM
What are the hematology effects antiphospholipid antibodies?
 
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Hematology: thrombophilia: Antiphospholipid antibodies: Antiphospholipid antibodies can be associated with an autoimmune hemolytic anemia and thrombocytopenia.
ARE THROMBOPHILIA STATES IN NON CIRRHOTIC PORTAL VEIN
 
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9th.World Comgress of the Intenational Hepato-Pancreato-Biliary Association-IHPBA-2010 - Buenos Aires Argentina -LIVER- DVD 5-FREE PAPER 5 Liver Surgery and Miscelaneous www.medicaldtv.com
Views: 211 medicaldtv
Bringing PCR diagnostics right to the point of care
 
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Fast, reliable flu diagnosis? Right at the point of care? Experts can’t believe how simple PCR diagnostics can be. For additional information, please visit : https://www.cobasliat.com/
How to Pronounce Diabetes Mellitus
 
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http://www.youtube.com/user/EnglishQ3
Views: 828 English
Lack of Sleep and Risk for Aggressive Breast Cancer
 
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Getting less than 6 hours a night of sleep is a risk factor in postmenopausal women with stage 1 or 2, estrogen positive, node negative breast cancer using the Oncotype DX tumor test. It measures the risk of tumor recurrence based on the expression of 21 oncogenes. Lack of sleep causes inflammation in the body that increases the risk for obesity, type 2 diabetes, heart attacks, strokes, osteoporosis, hypertension, cancer and much more. It results in both leptin and insulin resistance. Diet, exercise, weight management, stress reduction, adequate sleep, and avoidance of toxic exposures are all lifestyle factors that are important fundamentals that maintain good health. Take Dr. Saputo's Breast Cancer Health Assessment Once you have breast cancer you need to know the best strategies to slow down or reverse its growth. In Dr. Saputo's Breast Cancer Health Assessment we will ask you about the type of breast cancer you have, what treatment you've considered or completed, whether or not you're interested in CAM approaches to treatment, and important lifestyle factors that could have an impact on how your cancer will affect you. You will immediately be emailed back audios and videos Dr. Saputo believes will guide you to information that can help you manage your cancer and support the body's immune defenses. There is a lot we can do to prevent activating cancer genes and to protect us from developing breast cancer. Most breast cancers are epigenetic, not genetic! http://doctorsaputo.com/a/breast-cancer-health-assessment
Views: 369 DoctorSaputo
IEF Project Spotlight: Therapeutic Plasma Levels of Aminocaproic Acid in Asian Elephants
 
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IEF Project Spotlight: Therapeutic Plasma Levels of Aminocaproic Acid in Asian Elephants (Final Report) The goal of this project was to determine if aminocaproic acid (EACA), a drug used to prevent uncontrolled bleeding, could be effective in Asian elephants. Elephants can experience uncontrolled bleeding from a variety of causes, including following an infection with the elephant endotheliotropic herpesvirus (EEHV). Due to their large size and limited blood banking resources, treatment options for an elephant with uncontrolled bleeding are severely limited. Aminocaproic acid is widely used in human and domestic animal medicine to stabilize blood clots and prevent hemorrhage, and may have applications to elephants as well. We succeeded in developing an in vitro model of hyperfibrinolysis (blood clot breakdown), mimicking a diseased state. We then succeeded in reversing this diseased state through addition of EACA. The elephant plasma required a lower concentration of EACA to inhibit fibrinolysis, compared to humans and dogs. Our results suggest that EACA may be effective in preventing bleeding in elephants in vivo, and may be a valuable treatment option for a variety of bleeding disorders, including animals with EEHV infection presenting with hemorrhage. For more information about this project, and other projects to protect elephants, visit www.elephantconservation.org
Views: 193 IntElephantFound
Antiphospholipid Syndrome (clip)
 
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** To watch this video in full and claim a CPD credit please click here: http://www.rsmvideos.com/index.php/videoPlayer/?vid=319&class=videoThumbOdd Professor Anisur Rahman discusses Antiphospholipid Syndrome. He describes what the syndrome is and how to diagnose it. He also explains how to manage APS in pregnancy and suggests new tests and therapies for it. This Lecture was filmed at an event called 'Current theories and management of CTD' at the Royal Society of Medicine in London. Date of Lecture: 10th October 2012 The Royal Society of Medicine is an independent, apolitical organisation, founded over 200 years ago. We are one of the largest providers of continuing medical education in the UK.
Antiphospholipid syndrome Symptoms
 
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Antiphospholipid syndrome Symptoms
Views: 142 HealthSymptoms
Interventional Therapeutic Techniques in Acute Stroke
 
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The acute stroke webcast reviews the most current research and guidelines on time-based treatment of acute stroke and the use of tissue plasminogen activator. Visit http://www.ccfcme.org/strokevgrvideo to claim CME credit or learn more about the Stroke Virtual Grand Rounds series. The case-based stroke webcast features expert faculty member, Dr. M. Shazam Hussain, of Cleveland Clinic, and commentary by Activity Director, Dr. Edward Manno. The video was produced by the Cleveland Clinic Foundation Center for Continuing Education and the Cleveland Clinic Cerebrovascular Center. Interested in related CME education? Visit http://www.clevelandclinicmeded.com/specialties/Neurology.aspx?id=133&name=Neurology
Views: 345 ClevelandClinicCME
Health Alert: Greater risk of stroke seen in younger people
 
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CINCINNATI (Liz Bonis) -- May is stroke awareness month. A new report says there is a dangerous trend in those who are having the most common types of stroke or injury to the brain. Some alarming numbers are in this new report. Not just in the number of strokes people have each year, but those who do have strokes are at younger ages. One of the best ways to lower stroke risk is to reduce your blood pressure. Many people at the Mercy HealthPlex are in cardiac rehab which helps you recover from heart attack and stroke. But according to the new report from the American Academy of Neurology, researchers found about 15 percent of ischemic strokes,the most common type, are in those ages 18 to 44. They are not sure why more young adults are having stroke. It may be due to higher obesity rates, or that we don't watch blood pressure, and it may be due to diabetes rates also. We might think of these as minor inconveniences, but they can play a role in stroke risk. If you have these, ask your doctor how to lower your risk. Over time these things can make a difference. Follow Liz Bonis on Twitter @lbonis1 and LIKE her on Facebook.
Views: 87 LOCAL 12
EMT 5-11:  Obstetrics and Gynecology
 
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Module 5-11 of the Wisconsin 2012 EMT curriculum - Obstetrics and Gynecology.
Views: 59547 WCTCEMS
Factor 2 Mutation
 
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Views: 31 Lenora Castro
NCLEX / TISSUE PLASMINOGEN ACTIVATOR (tPA)/FIBRINOLYTIC /
 
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The video is about the use of tissue plasminogen activator , and it’s important , nclex style question which are frequently asks , one must know certain thing related to this . Hope this really help who are in need .
Views: 111 Pass Nclex rn
Pregnancy-Related Venous Thromboembolism [Hot Topic]
 
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Did you know that venous thromboembolism, or blood clots, is one of the leading causes of death among pregnant women? Fortunately, this condition can be successfully managed if identified and treated appropriately. In this month’s Hot Topic, my colleague, Dr. Ariela Marshall, will provide an overview of venous thromboembolism in the pregnant population, including its risk factors, treatment and prevention.
Endothel dysfuction and hypercoagulation state-hypercoagulation: How do they relate?
 
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Date : 13 September 2013 Speaker : Prof. dr. Teguh Asaad Suhatno Ranakusuma, SpS (K) Venue : ANVIN, Hotel Ritz Carlton Kuningan - Jakarta
Views: 87 Meetmed
Can brain tumors cause neuropathy ? | Best Health Channel
 
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Peripheral neuropathy (nerve damage) know the 10 most common symptoms of brain cancer. A small fraction of patients with. Both the 15 1999 metabolic encephalopathy is common nonmetastatic cause of altered mental diagnosis metastatic brain tumors can be confirmed by or neuropathy, caused dysfunction inferior alveolar nerve 23 2011 as most us know, a good scratch satisfy an itch. Weakness or numbness some brain tumors cause weakness on one side of the body other paraneoplastic syndromes, like peripheral neuropathy (numbness tingling in hands and feet) nerves are located outside spinal cord. Agents and radiation used to treat cancer also can cause peripheral neuropathy many cancers exhibit a propensity toward spread the cns, brain result in hypercoagulable state causing cerebrovascular complications. Sometimes, peripheral neuropathy can be caused by trauma or pressure on a nerve (such as from carpal tunnel syndrome) long term, heavy alcohol use 18 certain complications of cancer treatments cause and, patients with cancers the nervous system such brain tumors, 5 (medical xpress) researchers plymouth university peninsula schools medicine and dentistry are part an international team which nerves ones located outside your spinal cord. Brain cancer symptoms & signs brain. Different types, but peripheral neuropathy is the most common in those with cancer. Anxiety causes neurological symptoms calm clinic. Peripheral neuropathy symptoms, causes, and treatmentperipheral symptoms causes mayo clinic. Study finds potential prevention of common chemotherapy side effectans brain tumor center nj. Cancer neurological complications of systemic cancer american family brain neuropathy what is neuropathy? Definition & treatment. Md anderson cancer brain tumours and peripheral neuropathy medical xpress. Is when the nerve problem affects nerves outside of brain and spinal cord. Peripheral neuropathy in cancer patients. They are other types of cancer treatments can also cause peripheral neuropathy 11 certain chemotherapy drugs neuropathy, especially at higher doses or and, finally, tumors put pressure on nerves release 22 & neurological symptoms brain and cancers the cns significant morbidity. Brain cancer symptoms & signs brain nervous system side effects and spine intermountain healthcare. General radiation therapy to the brain and or spine can cause cns problems those most at risk are with diabetes, patients neuropathy should be tested for this. It can cause paralysis if a nerve is completely lacerated, although total controlling chronic condition not eliminate your neuropathy, but it caused by tumors and traumatic compressive conditions sometimes be treated surgically learn about the causes, symptoms, diagnosis & treatment of peripheral drugs, cancers, nutritional deficiencies, other disorders many causes acquired neuropathy include physical injury (trauma) to nerve, tumors, toxins, autoimmune responses, some scientists hope identify substances that will block brain chemicals generate pain signals, while hi there, several things, both dex temador weaknes what i have noticed de x tewill break down mussles make you 17 2011 among trauma, infection, susan's had all these at level since her tumor was discovered nerves are located in or spinal cord. What is peripheral neuropathy nccn. And peripheral nerves and chemotherapy induced neuropathy cranial is a disorder that causes nerve damage in the arise from are those directly your brain or brainstem but head injury, stroke, tumor can also cause fourth palsy spinal cord considered central nervous system. University of iowa peripheral neuropathy fact sheet. Peripheral neuropathy in persons with cancer. Eyesight after brain tumor restore vision. Optic neuritis risk factors, causes, and symptoms healthline. Googleusercontent search. Yet the it appears in many of same conditions that can cause chronic other spur neuropathic itch include spinal cord lesions, brain tumors, and 17 neuropathy also be classified according to disease causing. Relieving such symptoms of peripheral nerve changes (peripheral neuropathy). The common causes peripheral nerves send sensory information back to the brain and spinal cord, such as a less severe traumas also can cause serious nerve damage. Peripheral nerve disorders columbia neurosurgery. Polyneuropathy brain, spinal cord, and nerve disorders merck peripheral neuropathy brainfacts. National institute of neurological neurologic complications cancer and its treatment ncbi nih. Ctca cancercenter brain cancer symptoms url? Q webcache. Neuropathy is a disorder that causes nerve damage in the nerves arise from cranial are those directly your brain or brainstem and but head injury, stroke, tumor can also cause fourth palsy. They carry specific types of chemotherapy can injure peripheral nerves, particularly high dose. Many diseases and conditions can cause nerve damage. Tumors benign or malignant tumors of the nerves nearby 28 peripheral neuropathy can affect a r
MISS Symposium 2014
 
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Dr. Philip R. Schauer, Cleveland Clinic, invites you to the 2014 Minimally Invasive Surgery Symposium (MISS) February 26 - March 1 at the Venetian/Palazzo in Las Vegas. This year's MISS will focus on 4 main topics in advanced laparoscopic surgery: colon, hernia, foregut, and metabolic/bariatric.
Views: 178 MISSconference
Charité Clinical Journal Club by Fred Luft - 30.07.2014
 
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The N Engl J Med image of the week shows an extended tongue. You are offered amyloidosis, amyotrophic lateral sclerosis, pellagra, pernicious anemia, and Sjögren’s syndrome. All these conditions can involve the tongue. Antiphospholipid syndrome or antiphospholipid antibody syndrome (APS or APLS or) is an autoimmune, hypercoagulable state caused by antiphospholipid antibodies. APS provokes blood clots (thrombosis) in both arteries and veins as well as pregnancy-related complications such as miscarriage, stillbirth, preterm delivery, or severe preeclampsia. Renal failure commonly develops and transplantation is then necessary. The molecular pathways involved in the vasculopathy of the antiphospholipid syndrome are unknown and adequate therapies are lacking. Invesitgators used double immunostaining to evaluate pathway activation in the mammalian target of rapamycin complex (mTORC) and the nature of cell proliferation in the vessels of patients with primary or secondary antiphospholipid syndrome nephropathy. They also performed a brief intervention trial. This study opens up novel options for antiphospholipid antibody syndrome. The safety and effectiveness of automated glycemic management have not been tested in multiday studies under unrestricted outpatient conditions. Diabetologists conducted two random-order, crossover studies with similar but distinct designs, to compare glycemic control with a wearable, bihormonal, automated, “bionic” pancreas (bionic-pancreas period) with glycemic control with an insulin pump (control period) for 5 days in 20 adults and 32 adolescents with type 1 diabetes mellitus. Interestingly, the Model 4S iPhone played a pivotal role. Interleukin-17A is considered to be central to the pathogenesis of psoriasis. Investigators next evaluated secukinumab, a fully human anti-interleukin-17A monoclonal antibody, in patients with moderate-to-severe plaque psoriasis. The antibody was compared to etanercept therapy. Hematopoietic stem-cell transplantation is a potentially lifesaving therapy for several blood cancers and other diseases. For patients without a suitable related HLA-matched donor, unrelated-donor registries of adult volunteers and banked umbilical cord–blood units, such as the Be the Match Registry operated by the National Marrow Donor Program, provide potential sources of donors. The next investigators’ goal was to measure the likelihood of finding a suitable donor in the U.S. registry. Looks like it helps to be white and rich. Asplenia refers to the absence of normal spleen function and is associated with some serious infection risks. Hyposplenism is used to describe reduced ('hypo-') splenic functioning, but not as severely affected as with asplenism. We review the management of persons with poor or no splenic function. We then visit a 6 year-old girl with tinnitus in one ear. Our patient of the week is a 41 year-old man with fever leukopenia, lymphocytosis, abnormal liver enzymes, and a morbilliform rash. Etrolizumab is a humanized monoclonal antibody that selectively binds the β7 subunit of the heterodimeric integrins α4β7 and αEβ7. Lancet investigators aimed to assess etrolizumab in patients with moderately-to-severely active ulcerative colitis, compared to conventional therapy and placebo. Patients with radioactive iodine (131I)-refractory locally advanced or metastatic differentiated thyroid cancer have a poor prognosis because of the absence of effective treatment options. In the next study, investigators assessed the efficacy and safety of orally administered sorafenib in the treatment of patients with this type of cancer. Müllerian agenesis, also called Mayer-Rokitansky-Küster-Hauser syndrome or MRKH, named after August Franz Joseph Karl Mayer, Carl Freiherr von Rokitansky, Hermann Kuster, and G. A. Hauser, is a congenital malformation characterized by a failure of the Müllerian duct to develop, resulting in a missing uterus and variable malformations of the upper portion of the vagina. We next inspect an astounding study involving tissue engineering to help these women by constructing new vaginas and improving their quality of life. Autologous native cartilage from the nasal septum, ear, or rib is the standard material for surgical reconstruction of the nasal alar lobule after two-layer excision of non-melanoma skin cancer. The next tissue engineers assessed whether or not engineered autologous cartilage grafts allow safe and functional alar lobule restoration. These investigators also achieved a remarkable positive result. Pylephlebitis (also called infective suppurative thrombosis of the portal vein) is an uncommon thrombophlebitis of the portal vein or any of its branches. We review the topic.
Coagulopathies Part 2
 
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Views: 154 alphafag
Optimizing Outcomes of Peripheral Artery Interventions: The Role of Statins
 
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Jeffrey A. Southard, MD on the role of statins in peripheral artery interventions. Series: "Vascular Care 2011: Best Practices in Vascular Therapy" [12/2011] [Health and Medicine] [Professional Medical Education] [Show ID: 22934]
Views: 156 UCTVSeminars
GI Cops
 
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a short video clip about the impact of eating a unhealthy and high glycemic diet
Views: 316 sandyrolston
What are the diagnostic criteria for the antiphospholipid antibody syndrome?
 
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Hematology: Thrombophilia: Antiphospholipid Antibody Syndrome: Patients with the Antiphospholipid antibody syndrome must also have a positive IgG anticardiolipin antibody, a positive lupus anticoagulant, or both a positive IgM anticardiolipin antibody and a positive lupus anticoagulant test.
Charité Clinical Journal Club by Fred Luft - 26.09.2017
 
58:24
The N Engl J Med image of the week shows a head-and-neck CT of a 35 year-old man with neck pain and swallowing difficulties for over a year. You are offered cervical spondyloarthropathy, fish-bone foreign body, temporomandibular joint dysfunction, Ernest syndrome, and Eagle’s syndrome. Knowledge of head-and-neck anatomy is helpful here. Experimental and clinical data suggest that reducing inflammation without affecting lipid levels may reduce the risk of cardiovascular disease. Yet, the inflammatory hypothesis of atherothrombosis has remained unproved. Investigators conducted a randomized, double-blind trial of canakinumab, a therapeutic monoclonal antibody targeting interleukin-1β, involving 10,061 patients with previous myocardial infarction and a high-sensitivity C-reactive protein level of 2 mg or more per liter. The trial compared three doses of canakinumab (50 mg, 150 mg, and 300 mg, administered subcutaneously every 3 months) with placebo. The primary efficacy end point was nonfatal myocardial infarction, nonfatal stroke, or cardiovascular death. Canakinumab induced more sepsis deaths, which is not surprising. However, cancer deaths were reduced, which surprised me. Effects on cardiovascular outcomes was significant but at $200,000 per year hardly practicable. Heparin is used as the anticoagulant for percutaneous coronary interventions in Europe, while bivalirudin is favored in the USA. Swedish investigators enrolled patients with either ST-segment elevation myocardial infarction (STEMI) or non-STEMI (NSTEMI) who were undergoing PCI and receiving treatment with a potent P2Y12 inhibitor (ticagrelor, prasugrel, or cangrelor) without the planned use of glycoprotein IIb/IIIa inhibitors. The trialists found no significant difference between treatment with bivalirudin and treatment with heparin with respect to the rate of death, repeat myocardial infarction, or major bleeding events during 180 days of follow-up. US surgeons report 12-year outcomes for Roux-en-Y gastric bypass surgery for obesity. There are two control groups, namely the uninsured and the unwilling. Weight loss comprised about 30% of body mass in the operated, while the control groups lost no weight. Type-2 diabetes regressed, at least if the patients were not yet insulin-dependent and new cases of diabetes were decreased. A salubrious effect on blood pressure is also reported. We next inspect a genome-wide association study on gestational duration and preterm birth. Five genes were found with astoundingly low p values. However, the practicable magnitude of the effects was not given. There are excellent reviews on food allergy and the point of performing molecular diagnostic screening (thrombophilia testing) after venous thromboembolism. The case of the week involves a 59 year-old woman with a two week history of an asymmetrical polyarthropathy with acutely inflamed affected joints. Duchenne muscular dystrophy (DMD) is a severe, progressive, and rare neuromuscular, X-linked recessive disease. Dystrophin deficiency is the underlying cause of disease; therefore, mutation-specific therapies aimed at restoring dystrophin protein production are being explored. Investigators aimed to assess the efficacy and safety of ataluren in ambulatory boys with nonsense mutation DMD. Ataluren is a small molecule designed to cause stop codons to be skipped over during translation. I found the results disappointing and modest. Cabotegravir and rilpivirine are antiretroviral drugs in development as long-acting injectable formulations. The LATTE-2 study evaluated long-acting cabotegravir plus rilpivirine for maintenance of HIV-1 viral suppression through 96 weeks. The injectable strategy was effective. mRNA vaccines combine desirable immunological properties with an outstanding safety profile and the unmet flexibility of genetic vaccines. Based on in situ protein expression, mRNA vaccines are capable of inducing a balanced immune response comprising both cellular and humoral immunity while not subject to MHC haplotype restriction. We inspect a German mRNA vaccine in humans directed against rabies. Japan has entered the era of super-ageing and advanced health transition, which is increasingly putting pressure on the sustainability of its health system. The level and pace of this health transition might vary across regions within Japan and concern is growing about increasing regional variations in disease burden. If the poor Japanese would just stop smoking and embrace healthier life styles, their Global Burden of aging could get even worse. We inspect reviews on cholera and on hypothyroidism. We close the session with a patient who has mycosis fungoides. How is this T-cell lymphoma best treated?